Male sexual dysfunctions are a common category of diseases and they undoubtedly have a great impact on men’s self-confidence, self-esteem and quality of life. Common male sexual dysfunctions include hypoactive sexual desire, hypersexuality, erectile dysfunction, premature ejaculation, non-ejaculation and painful ejaculation. I. Erectile dysfunction (a) the classification of erectile dysfunction Erectile dysfunction was formerly known as impotence, because it does not explain the specific pathological mechanism and has a derogatory meaning for men, so it is now called erectile dysfunction (Erectile dysfunction, ED), the following will be abbreviated as its English abbreviation ED. ED can be classified as organic, psychological or mixed, the following table helps to determine the nature of ED 1, endocrine ED: endocrine ED accounts for 8% to 9% of all ED, its diagnosis is simple and reliable, clear pathogenesis, reasonable treatment and high cure rate. Such as hypogonadism and hyperprolactinemia are the causes of ED. 2, neurogenic ED: neurogenic E accounts for about 10% to 15% of organic ED. Diabetes, multiple sclerosis spinal cord injury and other disorders caused by neuropathological changes can lead to impairment of nerve conduction and prevent the transmission of nerve information to the smooth muscle of the penile corpus cavernosum. 3, vascular ED: vascular ED is the highest incidence of organic ED, which is divided into venous ED and arterial ED, is the main cause of secondary ED after the age of 40. The causes of venous ED include: the congenital presence of the cavernous body of the penis or acquired generation of too large a diameter or excessive number of veins; aging or unexplained degenerative changes in the white membrane; atrophy or fibrosis of the smooth muscle of the cavernous body; abnormal release of neurotransmitters. When the intracavernosal pressure is thus below 80 mm Hg, it is not possible to effectively reduce venous return, resulting in a weak erection or a short erection. If the aorta, iliac artery, pubic artery and internal pubic artery and its branches are blocked or abnormal development, it will prevent the flow of blood to the penis, thus leading to the occurrence of arterial ED. 4, drug ED: sleeping pills, sedatives, phenothiazines, antidepressants, antihypertensive drugs, anti-Parkinson’s drugs, etc. can lead to the occurrence of ED, to deal with the treatment of the primary disease and avoid drugs affecting erectile function. 5, psychological ED: psychological factors that cause ED include: due to the negative impact of the development process and trauma, so that they fear failure; they often lack self-esteem, self-confidence, full of inferiority complex or poor sense of body image; lack of communication between partners or lack of trust in the partner, or even with hatred; fear of the partner and fear of women; operating expectations are too high, excessive attention to sexual performance and even All of these factors can cause various types of emotional reactions such as anxiety and depression, which act on certain neurotransmitters in the higher and lower centers and ultimately affect the physiological function of the penis. Most psychogenic EDs still respond well to psychological and behavioral treatments and have a good prognosis. (ii) Introduction and evaluation of ED treatment methods The principles of selection as first-line drugs are: non-invasive; highly effective for patient-specific conditions; adequate sub-medical indications; minimal adverse effects and no contraindications; low cost; and easy to use. Oral medications are of course the first choice, especially those taken when needed. Viagra, or sildenafil citrate, enhances nitric oxide activation of cyclic guanosine synthesis by inhibiting phosphodiesterase type 5, which stimulates smooth muscle relaxation and causes erection. Prostaglandin E1 can directly stimulate smooth muscle relaxation and cause erection by activating adenylate cyclase, but it needs to be injected directly into the penile corpus cavernosum to be effective, and can also be administered via the urethra (Mythos) or topically (cream Bifadil). There are also foreign preparations of prostaglandin E1 and prazosin combination ointment, poppy berry gel, etc. The antidepressant Meprobamate has the effect of inhibiting 5hydroxytryptamine and promoting erection, and it can have 50% efficiency in treating psychogenic erectile dysfunction. Other treatment methods include negative pressure suction and narrowing methods, penile prosthesis implantation method. In short, ED is a common disease in men, once the man suffering from this sexual dysfunction should go to the regular hospital for medical attention, do not have a disease or blindly take the so-called aphrodisiacs. Second, premature ejaculation refers to persistent and repeated ejaculation under minimal sexual stimulation before, during or just after insertion, which is also against one’s own subjective will. We should make a comprehensive judgment in a holistic clinical context, without a one-sided emphasis on the number or duration of strokes or, more unconscionably, on whether the woman can achieve orgasm before ejaculation. Obviously, various organic factors such as excessive nerve conduction or abnormalities in neurotransmitter release can cause premature ejaculation; one argument is that it is due to prostatitis, but inflammation mostly comes and goes, whereas premature ejaculation is persistent, so this argument can be dismissed; anxiety, which is a common feature of almost all sexual dysfunctions, is certainly no exception as a cause of premature ejaculation; premature ejaculation is often the result of a critical period of sexual learning and a specific environmental Premature ejaculation is often the result of developing a habit of rapid ejaculation in the context of critical sexual learning and specific environments; partner tension also often causes premature ejaculation. Antidepressants are a new generation of ejaculation-delaying drugs that increase the level of 5-hydroxytryptamine (5-HT) in the body, and elevated levels of 5-HT are thought to be one of the mechanisms that inhibit ejaculation, such as fluoxetine, methocarbamol, chlorpromazine, sertraline and paroxetine, which have both anticholinergic effects. Third, non-ejaculation non-ejaculation patients in sexual life, the penis can be erect without difficulty, but also highly eager for the release of orgasm and receive sufficient effective sexual stimulation, but even if the intercourse for a long time, has made both sides feel very tired or even unpleasant, but still can not reach orgasm, the salient feature is that as long as the patient’s penis is located in the vagina can not ejaculate. From a clinical point of view, non-ejaculation and premature ejaculation are the two extremes of ejaculation disorders. Patients with ejaculation have an ejaculatory threshold that is so high that they are unable to ejaculate even with adequate and effective sexual stimulation, or even with super-intense sexual stimulation, whereas patients with premature ejaculation have an ejaculatory threshold that is too low. It is now believed that most cases of ejaculation are psychological or situational in nature because they are able to ejaculate during sleep or during masturbation, but only during intercourse. When a man never ejaculates in any situation, he has a high probability of organic impairment and they account for approximately 10% of cases of non-ejaculation. Neurological factors include both central and peripheral. Central refers to the absence of orgasm or ejaculatory movements in patients due to abnormal brain function and insufficient production of peripheral stimuli or failure to transmit to the center, and therefore failure to excite the ejaculatory center or enhanced inhibition of the ejaculatory center. Peripheral refers to damage to the nerve during surgery, pathological changes in the nerve itself, or the effect of anti-sympathetic drugs on nerve endings or receptors can cause damage to the peripheral efferent nerve, sometimes resulting in permanent loss of the patient’s ability to ejaculate. Drugs with anti-sympathetic activity for hypertension, neurological disorders (e.g., depression), and psychiatric disorders often have adverse effects that impair ejaculation, such as reduced semen volume or absence of ejaculation during orgasm in half of the patients taking methiodarazine. Ejaculation can be induced with commercially available electric massagers of various types, regardless of psychological or organic ejaculation, often with good results. At first it may take 10 to 15 minutes of continuous stimulation, but later it only takes 5 minutes to achieve the purpose of ejaculation. The stimulation area is mainly at the glans and tether, but can also be moved up and down along the penile shaft. Sometimes it takes more than ten treatments to heal. Symptomatic treatment includes oral ephedrine, circumcision, cessation of smoking and alcohol, improvement of the living environment, and strengthening of physical fitness.