Infectious eczema-like dermatitis is a limited acute eczema-like dermatitis that occurs on the basis of an infectious lesion. It spreads gradually to the vicinity, often in patches of oozing wetness and crusting, and can also be dry and flaky. The diagnosis is based on the fact that the disease is based on an infected lesion with vesicles in the surrounding skin. Acute eczema-like lesions such as blisters and pustules are not difficult to diagnose, but should be distinguished from contact dermatitis, eczema, impetigo and seborrheic dermatitis. Treatment measures first use antibiotics to clear the primary infected lesions, antibiotic sensitivity tests can be done in order to select sensitive antibiotics. Corticosteroids such as prednisone and dicamisone can be used to rapidly reduce severe acute inflammation and to stop inappropriate topical medications. Local in the acute exudative state, available 1:5000 ~ 1:8000 potassium permanganate solution, 1:20 compound aluminum sulfate solution (Bro solution) wet compresses. When the exudate is reduced, topical antibiotic solutions such as 1% gentian violet solution, gentamicin, lotions or emulsions can be used. In the state of chronic dermatitis with little exudate, topical application of 10% ichthyolite ointment, Bactrim ointment, as well as erythromycin and chloramphenicol ointment can be used. Etiology The patient first has a local staphylococcal infection, and the disease spreads from this infected lesion to the nearby expansion, not only as a self-inoculation process but also as a manifestation of autosensitivity. Infected lesions can be classified as otitis media, boils, carbuncles, ocular, ear or vaginal discharges, traumatic infections, and chronic ulcer and burn infections. Eczema-like dermatitis may develop in the nearby skin as a result of an allergic reaction to bacterial or other products of the exudate from the lesion or to the damaged tissue. The primary focus may be a blister or pustule, a scaly or crusted inflamed papule, or a moist red spot, often occurring symmetrically over an exposed area. Sometimes the earliest damage is a festering abscess, boil, carbuncle, sinusitis, chronic otitis media, decubitus ulcer, fistula, scabies, or ulcer, or it can be a focal infection of the nose, eye, or vagina. Often, as the local lesion worsens, the skin around the infected site develops erythema, papules, blisters, pustules, and crusts, and gradually spreads outward into a patchy eczema-like dermatitis exuding pulpy pus with a crusty surface. When the symptoms are mild or when the inflammation subsides, the affected area is dry and flaky. In severe cases, the affected area can be swollen, vesiculated, exudate, and have obvious acute eczematous changes such as erythema, papules, small blisters, and pustules on the periphery, becoming eczematous patchy skin damage with irregular borders. Local lymph nodes are often enlarged, and there is occasionally an increase in body temperature. Extensive dermatitis may also occur elsewhere on the body as a result of self-sensitivity reactions. Some patients often develop linear or striated eczematous dermatitis due to scratching, and the rash is often asymmetric with intense itching.