There is no doubt that diabetes is characterized by high blood sugar. However, hyperglycemia is not the only thing that causes its complications. For, there is another side to hyperglycemia, such as the failure of timely breakdown of blood sugar or impaired and delayed energy production of sugar. The former mainly leads to the metabolism of sugars by adiponectin to sustain life, resulting in increased lipolysis and protein consumption. The latter mainly leads to increased fat synthesis and the process forms complications. Because, the delayed energy production of sugar, the energy required for life has been provided by adiponectin and therefore only saturated fat can be synthesized. At the same time, with the appearance of urine sugar, sugar energy is lost, and the energy needed to maintain life requires more from fatty proteins, and because the blood glucose supply will also be inadequate, it will be necessary to protein and glycerol-based isogenic blood glucose in fat, otherwise life cannot be maintained. Therefore, not only will the patient’s blood sugar continue to rise, but more fat will enter the bloodstream and more protein will be consumed. At the same time, the loss of glycerol groups from fat due to xenobiotic blood glucose results in the release of more harmful low-density and free fat into the bloodstream. Thus, it is not only the high blood sugar itself that leads to complications of diabetes, but also the increased frequency of fat metabolism leading to reduced efficiency and continued insulin insensitivity, initial hyperactivity, and subsequent failure. As well as the double hyperactivity of lipolysis and anabolism, protein overconsumption, etc., formed by the increase of fat and protein in place of sugar production for life support and participation in gluconeogenesis due to impaired glucose metabolism and its loss in urine. Among them, hyperglycemia mainly leads to microvascular and its abundant organs fundus and kidney damage; abnormal functions of peripheral nerves and its abundant organs skin, gastrointestinal and bladder; elevated blood osmotic pressure and its water and electrolyte metabolism disorders. Fat catabolism and anabolic double hyperactivity mainly lead to blockage of large blood vessels such as heart and brain limbs, forming a series of complications such as cardiovascular and cerebrovascular diseases, diabetic foot, etc., and lead to ketoacidosis, etc. in the case of relatively insufficient sugar catabolism, which cannot effectively help metabolize increased fat to burn completely; protein overconsumption mainly leads to lower immunity and the body’s own repair ability, combined with wounds that do not grow, infections that are difficult to heal, anemia, tumors, etc. Other diseases. However, the above process exercise drugs can never stop independently, and without a proper diet as a basis, these treatments are themselves etiological complications leading to factors. Because, there is no technology that can replace food to sustain life, including the food sugars. Or although the basic pathology of diabetes is characterized by impaired pancreatic islet function and relative and absolute insulin deficiency, its dietary etiology and the vast majority of its complications occur entirely in the process of eating energy to save, not eating or not eating enough to require reserves and their nutritional interchange to sustain life. For example, exercise can promote muscle without insulin to lower sugar directly, but only through consumption. And at the same time consume fat protein, so it will put more demands on the diet. Otherwise, not only the consumed fat will come back, directly leading to protein deficiency, and lead to the need for more sugar requirements. Failure to meet this will again affect the normal metabolism of protein and fat, and the process will cause new problems. Drug hypoglycemia is similar to this. For example, insulin and its pro-secretion and sensitization help blood glucose decomposition, which cannot prevent excessive sugar intake and cannot consume synthetic fat after decomposition; α-glucosidase inhibitors inhibit food sugar decomposition and absorption, and metformin and other drugs promote direct metabolism of blood glucose in peripheral tissues, which can only mimic the direct pathology of diabetes glucose metabolism disorder under conditions of low or insufficient sugar intake, leading to more production capacity of adiponectin and its greater participation in gluconeogenesis . The process is also a source of aggravating adiponectin metabolism disorders. Among other things, increased fat metabolism, aggravating the blockage of large blood vessels such as the heart, brain and limbs, protein consumption can not be replaced in a timely manner, reducing the body’s immune and self-tissue repair capacity. Therefore, the management goals recommended by diabetic dietary guidelines around the world are to meet ideal body weight with total energy, reduce total fat metabolism and its saturated fat energy ratio, and at the same time have a slightly higher proportion of protein than normal and a slightly lower proportion of sugar than normal. That is: total caloric energy to maintain the ideal body weight. Among them, protein accounts for 15-20% of total caloric energy, more than half of high-quality protein; 55-60% of carbohydrates; 25-30% of fat, saturated, mono- and polyunsaturated fatty acids combined capacity ratio 1:1:1. Because, not only dietary energy with excess fat or obesity is the basic cause of type 2 diabetes, any other type of diabetes superimposed etiological factors, and complications are not only high blood sugar. Rather, at the same time there are exercise drugs can not be independently corrected, the absence of a reasonable source of diet will also lead to with aggravation of the sugar production capacity is insufficient, too much, etc. caused by fat decomposition anabolic double hyperactivity, protein is overconsumed, etc. That is: its nutritional management goals are formulated according to the physiopathological characteristics of diabetes. However, since the patient’s diet is currently out of control. Therefore, naturally any glucose- and lipid-lowering techniques are available, but the proportion of patients who die due to their cardiovascular complications is as high as 50%-80%, which is four times higher than that of non-diabetes; the proportion of patients who die due to cerebrovascular complications is as high as 10%-15%, which is twice as high as that of non-diabetes. At the same time, protein overconsumption leads to a decrease in autoimmunity and self-healing ability, which is not only an important cause of a large number of combined malignant infections, chest and ascites and other serious diseases in the late stages of diabetes, but also the occurrence of tumors is 2-3 times higher than that of non-diabetes. For example, some of the most scientific, authoritative and technologically advanced international evidence-based studies on diabetes, such as the American Action to Control Cardiovascular Risk in Diabetes (ACCORD), the United Kingdom Prospective Diabetes Study (UKPDS) and the study on the relationship between intensive glucose lowering and microvascular and macrovascular complications (ADVANCE), have all simultaneously encountered patients with insulin application Weight gain, obesity, aggravation of hyperlipidemia, etc., and problems related to the risk of complications due to the aggravation of the etiology of the drug caused by the irrational diet. At the same time, what lipid-lowering techniques are available, coronary heart disease human death first. Because, the drug originally could never independently correct the nutritional metabolism disorder. Therefore, help diabetic patients to effectively manage diet, not only conducive to lifting the diet etiology, crack diabetes rehabilitation, cure difficulties, protect the results of exercise drug prevention and treatment, natural reduction of complications, but also the basic safety premise of exercise drug help without adding to the chaos.