Diabetic complications are common chronic complications that are transformed by diabetic lesions that can involve one organ or multiple organs and are the leading cause of death in diabetic patients. Diabetic complications include: ketoacidosis, non-ketotic hyperosmolar coma, lactic acidosis, heart disease, cerebrovascular disease, extremity gangrene, neuropathy, nephropathy, retinopathy, and multiple infections caused by diabetes. The majority of diabetic complications are cardiovascular complications, including macroangiopathy, such as heart disease, hypertension, and cerebrovascular accidents, and microangiopathy, such as retinopathy and nephropathy. Other chronic complications include neuropathy, diabetic foot, and various infections caused by diabetes. Chronic complications of diabetes are the main cause of death in patients. The reason why diabetes is prone to infections is that high blood sugar in the patient’s body is conducive to bacterial multiplication, the ability of white blood cells to phagocytose bacteria is reduced, and the patient’s resistance to infection is reduced, resulting in urinary tract infections, whistling tract infections, and skin infections. Hyperglycemia also predisposes nerve cells and nerve fibers to lesions, causing some patients to develop symptoms such as diarrhea, constipation, urinary retention, and impotence. In addition, insufficient blood supply to the lower limbs and bacterial infection due to peripheral neuropathy can cause foot pain, ulcers, gangrene of the extremities, and other lesions called diabetic foot. Diabetes is so terrible, but it is not without medicine. Nowadays, with the advancement of medical technology, it is no longer necessary to control diabetes through medical treatment all year round, and it is entirely possible to try to treat it through surgical procedures. For example, gastric bypass surgery is a surgical procedure that reduces the volume of the stomach, shuts down most of its functions, and changes the structure of the intestinal tract, thus further reducing digestive capacity and thus controlling absorption. This is because the reconstruction of the gastrointestinal tract alters the secretion of hormones in the intestine-islet axis, thereby improving glucose metabolism.