The general management of patients with PCOS is based on dietary modification and weight control. A decrease in body mass index (BMI) improves fertility outcomes, suggesting that weight loss can affect reproductive endocrinology. Weight loss in obese, hyperandrogenemic, anovulatory women has been found to reduce insulin and free testosterone, while increasing SHBG. Studies of the relationship between hyperinsulinemia (fasting and postprandial) and anovulation have also shown that lower insulin levels are responsible for the recovery of ovulation. Ovulation outcome is correlated with BMI, suggesting that weight can influence pregnancy outcome. Weight control has implications for both fertility and severe metabolic disorders. Exercise as a method of weight loss results in lower insulin concentrations through glucose utilization by peripheral tissues. In addition, low calorie food intake reduces insulin secretion. Reducing body weight can reduce insulin concentration in blood and increase the concentration of sex hormone binding globulin and insulin-like growth factor binding protein, resulting in a decrease in ovarian androgen secretion and a decrease in free testosterone in blood. The basic method to improve high androgen in PCOS is through diet regulation and exercise. In conclusion, performing daily tasks or engaging in regular exercise or weight loss training may be beneficial for long-term physical health, ovulation and pregnancy. Good eating habits and exercise can promote weight loss, which can lead to higher pregnancy rates and lower treatment costs, and is a simple treatment for low fertility.