A burst fracture is a simple orbital trauma condition with orbital wall fracture, intact orbital rim, soft tissue impaction and typical clinical symptoms resulting from orbital contusion. Mechanism of occurrence: Mostly due to car accidents, sports and fights. The mechanism of occurrence is currently considered to be a sudden increase in intraorbital pressure caused by an external force acting on the soft tissues of the anterior orbit, which is partially absorbed by the intraorbital soft tissues and leads to fracture of the weakest part of the orbital wall according to the principle of hydraulic transmission. The orbital wall has the thinnest sieve cardboard and the thinnest posterior part of the infraorbital sulcus, which are the areas prone to fracture. The soft tissues are embedded in the fracture or in the maxillary or septal sinus due to the simultaneous action of fluid pressure on the soft tissues. Clinical manifestations: immediately after the fracture, symptoms such as protrusion of the eyeball, swelling of the eyelid, loss of vision and subcutaneous bleeding of the eyelid may occur. 1 week later, the edema disappears and the eye becomes sunken, diplopia and numbness of the affected side appears. Diagnosis: 1. History of oculofacial contusion; 2. Typical diplopia and entropion; 3. CT examination Routine examination should be done in both horizontal and coronal directions to observe the inner and outer walls of the orbit, as well as the upper and lower walls and adjacent soft tissues. Treatment: 1.Conservative treatment (1) The patient can accept the appearance of the eye within 2 mm (2) The degree of diplopia does not affect normal life; (3) The fracture area is small and there is no obvious soft tissue prolapse and embolism should be considered conservative treatment. 2.Surgical treatment (1) unrelieved diplopia; (2) obvious intraocular invagination; (3) CT shows muscle embolism should be performed surgically. Anesthesia: General anesthesia is usually used because of the depth of the surgical site. Instruments to be requested from the operating room: cerebral pressure plate, stripper, suction, mono- or bipolar electrocoagulation. Problems to be explained to the patient before the operation: short period of diplopia may occur due to the length of nerve entrapment before the operation, and eye movement should be relieved immediately; facial numbness may occur after the operation, which is normal; whether the correction of eye entrapment is satisfactory after the operation. Postoperative issues to be noted: postoperative infusion of antimicrobial + Demi 10 mg for 3 days; postoperative eye and orbital area should be pressure bandaged for at least 2 days, and daily testing for light sensation at the eye suture is required during the two days; skin sutures should be removed 5 to 7 days after surgery.