Cerebral hemorrhage is a common disease in neurological surgery and can be divided into hypertensive hemorrhage and non-hypertensive hemorrhage according to blood pressure. Hypertensive cerebral hemorrhage accounts for about 50% of all cerebral hemorrhages and even 70% to 90% of spontaneous non-traumatic cerebral hemorrhages in adults, and is a chronic pathological change of small blood vessels in the brain parenchyma, including (1) microaneurysms; (2) fatty hyaline changes; (3) both. Non-hypertensive cerebral hemorrhage accounts for about 25% to 50% of cerebral hemorrhage, caused by: (1) cerebral amyloid angiopathy; (2) vascular malformation: arteriovenous malformation is more common in young people, especially in women, and hemorrhage is located in the cerebral lobe (cortical or subcortical, because the malformed vessels are more superficial), and less amount is more considered. (3) Brain tumor: The causes of hemorrhage are spontaneous necrosis of tumor, rupture of tumor vessels, and tumor infiltration of vessel wall. The following conditions should be considered: a. Chronic headache and/or focal neurological signs before the disease; and/or psychiatric abnormalities and personality changes; b. Optic papillary edema at the onset of cerebral hemorrhage; c. The site of cerebral hemorrhage is not the typical site of so-called “primary” hemorrhage; d. Simultaneous hemorrhage in multiple sites; e. CT shows a central low-density ring-shaped hemorrhage (as a primary hemorrhage). e. CT shows a low-density circumferential hemorrhage (hemorrhage on the surface of the tumor) or irregular mixed density; f. Perifocal edema and occupying effect disproportionate to the hematoma; g. Enhanced nodular shadow around the acute hemorrhage foci after contrast injection. (4) Anticoagulation: factors predisposing to cerebral hemorrhage during anticoagulation: advanced age; large infarct size; high blood pressure; embolic infarction. Hemorrhage may occur alone, not necessarily combined with systemic hemorrhage; slow progressive course of hemorrhage; poor prognosis. (5) vasculitis; (6) pharmacologic, drugs that can cause elevated blood pressure or vascular hypersensitivity are possible, such as cocaine, amphetamines; (7) trauma.