Periodontitis makes coronary heart disease high Periodontal infections can cause acute or subacute infective endocarditis. It has been reported that 10% to 30% of endocarditis is associated with odontogenic infections or dental treatment. Severe periodontal disease can lead to an increased incidence of coronary heart disease. Endodontic infection and periodontitis are also independent risk factors for atherosclerosis, acute myocardial infarction, and especially ischemic stroke. In a large number of long-term follow-up surveys, it was found that patients with periodontitis had a 25% higher incidence of death or hospital admission due to coronary heart disease than those without periodontitis; other scholars reported that patients with periodontitis were 1.4 times more likely to develop coronary heart disease than those with normal periodontium. Patients with diabetes are more likely to develop periodontal disease and have a more severe incidence. Also, diabetic patients with periodontitis can have a significant decrease in blood glucose levels after periodontitis is effectively controlled. With poorly controlled blood glucose metabolism, diabetic patients are prone to periodontal tissue infections, which can further increase the difficulty of controlling blood glucose levels. If periodontal infections are actively and effectively controlled, maintaining healthy periodontal tissues can significantly improve individual glucose metabolic control and reduce insulin dosage; at the same time, control of periodontal infections plays a significant role in maintaining long-term metabolic levels in diabetic patients. Periodontitis increases the risk of slow-onset lung Many studies have found a correlation between periodontal disease and slow-onset lung. Epidemiological findings show that the incidence of respiratory disease is 1.3 times higher in those with poor oral hygiene than in those with good oral hygiene, and periodontitis may increase the risk of developing slow-onset lung. Dental plaque may be a host for respiratory pathogenic bacteria. H. pylori is hidden in dental plaque H. pylori is an important causative agent of chronic gastritis, peptic ulcers and other diseases, and is closely related to the occurrence of gastric cancer and gastric mucosa-associated lymphoma. The oral cavity may be a site for H. pylori, and the detection rate of H. pylori in plaque is significantly higher in patients with periodontitis than in those with healthy periodontium. The detection rate of H. pylori in subgingival plaque decreased significantly after basic periodontal treatment, and the eradication rate of H. pylori was significantly higher in patients with gastritis than in those who did not undergo basic periodontal treatment. Periodontitis is more severe in rheumatoid patients The prevalence and severity of periodontitis is higher in people with rheumatoid arthritis than in people without rheumatoid arthritis, and the prevalence of rheumatoid arthritis is higher in people with periodontitis than in the general population, but this correlation is not a cause-and-effect relationship. The prevalence of both periodontitis and osteoporosis increases with age. Systemic osteoporosis causes bone loss in the jaw, alveolar bone atrophy, and accelerated tooth loss in patients with periodontitis.