Insufficient iron stores at birth, dietary iron deficiency, and excessive growth and development of adolescent children cause iron deficiency in the body, resulting in reduced hemoglobin synthesis and causing iron deficiency anemia in children, which is the most common nutritional anemia in children. The disease develops slowly and is clinically characterized by small cell hypochromic anemia, reduced serum ferritin and effective iron therapy. The symptoms of iron deficiency in babies are mainly some manifestations related to anemia and anemia, starting with frequent irritability, mental inactivity, loss of appetite, and pale skin and mucous membranes, with the lips of the mouth, oral mucosa, nail bed and palms of the hands being the most obvious. Preschool and school-age children may report fatigue and weakness, dizziness, blackness before the eyes, and tinnitus. As a result of extramedullary hematopoietic reaction, the liver, spleen and lymph nodes are often mildly enlarged, and the younger the age, the more severe the anemia, and the longer the course of the disease, the more pronounced the enlargement of the liver and spleen. In addition, neuropsychiatric changes are also evident. Before the appearance of anemia, due to the decrease of ferritin, babies tend to be irritable and uninterested in their surroundings, and developmental assessment reveals that babies have poor concentration, reduced comprehension and slow reaction. Digestive system involvement may show a reduced appetite. Anemia can cause reduced immune function and is often prone to co-infections, repeated upper respiratory tract infections and digestive tract infections. Combined respiratory tract infections can also induce heart failure due to increased cardiac burden. Anthelmintic may occur due to abnormal epithelial tissue.