It is now well established that endometriosis is closely associated with infertility. The infertility rate in patients with this disease is 40%. I. It involves the effect of ectopic disease on the fallopian tubes. Ectopic lesions can affect the function of the fallopian tubes, manifesting tubal hypoplasia or obstruction. This tubal abnormality can significantly reduce the pregnancy rate in patients with ectopic disease. However, there is no significant correlation between the degree of ectopic lesions and the degree of tubal patency, because in some cases of moderate or severe lesions, adhesions around the fallopian tubes and restricted peristalsis significantly reduce the ability to conceive, but the effect on the lumen of the fallopian tubes is small, so this is one of the reasons why some patients are infertile even though their fallopian tubes are patent. Second, abnormal ovarian function leads to ovulation disorders and poor corpus luteum formation and reduces the conception rate. In ectopic patients, changes in the pelvic microenvironment such as local accumulation of PG (prostaglandins) and high local PG content in the fallopian tubes or imbalance in the ratio may cause abnormal peristalsis of the fallopian tubes, thus affecting egg collection, fertilization and fertilized egg delivery and resulting in infertility. Fourth, abnormal immune function leads to an increase in anti-endometrial antibodies that disrupt the normal metabolism and physiological function of the endometrium and affect the fertilization of eggs.