Congenital dislocation of the hip itself cannot cause femoral head necrosis, but either manual or incisional revision is prone to femoral head necrosis, a complication that results from varying degrees of damage to the blood flow of the femoral head during the resetting process. Another cause of femoral head necrosis is the need for external fixation after resetting. When the hip is fixed in an overflexed external or internal rotation position, the medial femoral artery is compressed, thus affecting the blood supply to the femoral head and neck. Moreover, maintaining the position for a long time may also increase the intraosseous pressure and cause venous reflux obstruction, resulting in venous stasis, further increasing the intraosseous pressure, decreasing the blood input, or even interrupting the blood flow, resulting in ischemic necrosis. Non-surgical treatment has a lower rate of recurrent necrosis compared to surgery. Preoperatively there is also the lowest rate of necrosis with more than two weeks of bone traction before resetting. If femoral head necrosis occurs after the femoral head bone appears, the head density will increase and appear flattened, and later there will be a mismatch of head and socket, a subluxation, and also a series of other sequelae, then certain orthopedic surgery is required for repair.