1, postoperative hematoma is a more common complication, if not handled in time can lead to flap skin necrosis, incision infection and delayed healing and other serious consequences. The causes of hematoma are incomplete intraoperative hemostasis, poor hemostasis by electrocoagulation, dislodgement of the ligature; poor postoperative negative pressure drainage and poor fixation by pressure bandaging; excessive postoperative upper limb activity, poor fitting of the flap to the wound, premature removal of the drainage strip, etc. After the occurrence of hematoma, there is usually obvious swelling and pain, bruising of the skin around the operation area, subcutaneous swelling and fullness can be seen in larger hematomas, and fluctuating sensation can be palpated, and bloody fluid or jelly-like blood clot can be extracted by puncture, and the largest hematoma in this group is about 400 ml of blood clot and bloody fluid removed. The hematoma can increase local tension, compress blood vessels and affect skin blood flow. The toxin produced by the hematoma can cause skin vasospasm, endanger blood flow and cause distal necrosis of the flap. The hematoma should be removed within 12 hours of formation to save the flap. Therefore, the hematoma should be treated promptly. If the amount of accumulated blood is small, a syringe can be used to draw the accumulated blood cleanly and then apply pressure bandage after negative pressure drainage; if the amount of accumulated blood is large or a large blood clot is formed, the incision should be opened to stop the bleeding completely, remove the stagnant blood clot, re-suture after careful examination to make sure there is no active bleeding point, apply negative pressure drainage, apply pressure bandage to fix, and give hemostatic drugs and dexamethasone static point treatment. 2, subcutaneous effusion usually occurs several days after the drainage tube is removed, the skin and the trauma surface are not close, the upper limb activity is too much, the subcutaneous continued to exude fluid to form subcutaneous effusion, there is a sense of fluctuation under the skin when touched. After aspiration, a yellowish serum-like fluid can be seen. Most of the causes are the existence of cavity between flap and subcutaneous tissue in the early postoperative period, necrosis and liquefaction of hair follicles or electrocoagulated sweat glands and fat remaining under the flap, premature withdrawal of drainage tube, and excessive exudate that cannot be absorbed. To prevent the occurrence of effusion, avoid excessive coagulation of adipose tissue by radiofrequency electrocoagulation during the operation, and the subcutaneous should be repeatedly rinsed after the operation. If fluid accumulation occurs under the skin, it can be healed by syringe aspiration followed by pressure bandage fixation for several days. 3, skin necrosis: is the most serious complication after axillary odor surgery. Mainly superficial skin trimming is too thin, the dermis and subdermal vascular network destruction, improper postoperative bandage fixation caused. In the early stage, pale white or dark brown and gray-black spots appear on the skin of the operated area, and black crusts are formed on the necrotic skin a week later. There are two sources of local skin blood supply after subcutaneous trimming: one is the residual dermis and subdermal vascular network; the other is the skin is attached to the trauma surface and can obtain nutrients directly from the trauma surface. Therefore, if intraoperative superficial trimming is too thin and destroys the dermis and subdermal vascular network, the skin should be sutured several stitches outside the stripped surgical area after surgery, and the pack pile should be fixed in order to make the skin and trabecular apposition close and firm, and the pack pile should be opened after 5-7. Intraoperative electrocoagulation should be distinguished between deep and superficial layers, and shallow electrocoagulation should be avoided as much as possible to burn the skin to avoid postoperative skin necrosis. Small areas of skin necrosis, generally less than 1 cm in width, can be healed by multiple drug changes; if large areas of skin necrosis occur, they should be treated promptly with skin implants or local flap repair after removal of necrotic tissue. For cases with incomplete treatment in the past for re-repair, due to unfavorable factors such as unclear subcutaneous anatomical levels and scar adhesions, subcutaneous trimming should be noted that if the dermal vascular network blood supply has been destroyed, postoperative piles of fixed skin pieces must be packed to prevent postoperative skin necrosis. 4.Infection: Infection is mostly seen after complications such as hematoma, subcutaneous fluid, skin necrosis, incision splitting. If incision infection is found, it should be treated by changing medication in time, and the medication should be changed diligently in hot weather. In order to prevent infection, the skin should be cleaned before surgery, the operation should be strictly aseptic, the incision should be observed regularly after surgery, and complications should be dealt with in a timely manner. 5. Incisional dehiscence and delayed healing: mostly seen after complications such as postoperative hematoma, subcutaneous effusion, incisional infection, skin necrosis, or incisional dehiscence after strenuous activity of the upper limb. Therefore, active prevention and treatment of complications as well as upper limb braking can prevent the incision from splitting and promote skin healing. 6, odor residue: mainly due to incomplete removal of the parietal sweat glands and insufficient scope and level of removal. Pathological histological observation shows that there is a hair follicle-sebaceous gland complex in the deep dermis of the axilla, and there are ducts and glands of the acrosomal sweat glands in the deep dermis and subcutaneously. The apocrine sweat glands are slightly deeper than the sebaceous glands, mostly at the junction between the deep dermis and the subcutis, but also in the subcutaneous fat layer, mostly just below the sebaceous glands. The range of surgical removal is generally about 1cm outside the axillary hair, but some patients exceed this range, and some patients have residual glands because the skin flap is trimmed too thickly during the operator’s operation. 7. Incision scar and scar contracture: the normal healing incision scar is not obvious, and the scar is less obvious after the incision split and the delayed healing incision heals with drug change treatment. However, scar adhesions were formed between the skin and the trauma surface in the operated area, and the upper limb lifting was partially restricted, probably due to the patient’s less fatty tissue in the axilla and scar contracture caused by the adhesions between the skin and the deep fascial tissue after surgery. Without any treatment, the symptoms will disappear on their own after the softening and relaxation of the scar after six months to a year. 8, skin petechiae: postoperative skin bruises around the area, mainly due to too tight postoperative bandages, poor skin circulation at the bandage site; subcutaneous trauma hematoma, the surrounding skin can also appear petechiae. Therefore, the pressure of bandage fixation should be appropriate, and skin bruises generally do not need to be treated; for bruises caused by hematoma, the bruises can be absorbed by themselves after the hematoma is cleared. 9. Axillary hair loss: 317 patients were observed to have lost almost all of their axillary hair six months after surgery, and a small number of patients had a little axillary hair present, but the axilla was odorless.