For cerebral vasospasm, it is mainly a change in the nature of the narrowing of the cerebral blood vessels, resulting in an ischemic change in the terminal blood supply area. Therefore, the most important clinical symptoms are mainly cerebral infarction, that is, ischemic necrosis of nerves. The first is to remove risk factors, including blood pressure control, blood sugar stabilization, smoking and alcohol cessation, to prevent further vascular deterioration. The second is to apply relevant drugs for treatment, generally in clinical practice is mostly based on nimodipine injection, occasionally can also apply fasudil for treatment, and this time if faced with an increase in blood pressure, can apply calcium antagonists, such as nifedipine. The third is to improve the intravascular osmotic pressure, basically for patients who can take orally to enhance nutritional therapy, if not oral patients can be treated with intravenous infusion of human albumin. The fourth is to actively treat the primary disease, such as cerebral vasospasm caused by subarachnoid hemorrhage, which can be treated by lumbar puncture to release the blood cerebrospinal fluid. In addition, it is important to prevent cerebral vasospasm in a timely manner.