Ventral cleft is a rare malformation caused by the prolapse of internal organs due to the absence of some of the constituent components of the abdominal wall next to the umbilicus. Ventral cleft was named in 1953 by Moore et al. based on its pathological features. Ventral cleft is less common than umbilical bulge and is only 1/5-1/10 of umbilical bulge, more frequent in male than female infants, 2:1 or 3:2, and half of them occur in preterm infants. The umbilicus is normally positioned and the umbilicus and cord morphology are normal in children with abdominal cleft. The abdominal wall cleft can be located on the left or right side of the umbilicus, with the majority (80%) on the right side, and the cleft is longitudinal, usually only 2-3 cm, and large clefts are rarely seen. There is a normal skin between the abdominal wall fissure and the umbilicus that is about 1-2 cm wide, and there are some extremely narrow ones that are not recognizable. The abdominal wall fissure lacks muscle and peritoneum. The prolapsed contents are the prointestine, from the stomach to the colon, and very rarely the bladder and uterus are visible. The intestinal canal is thick, hypertrophic and edematous, with adhesions between intestinal collaterals and marked shortening of the intestinal canal, which may be covered by a jelly foreign substance or a fibrinous pseudomembrane of meconium color. In cases of late presentation, there may be necrosis of the detached intestinal canal. If the intestinal canal is exposed outside the abdominal cavity immediately after delivery, the intestinal canal should be wrapped in saline antibiotic gauze and then wrapped in rubber gloves or plastic bags outside, and emergency surgery should be performed within a few hours of delivery.