Brown-Séquard syndrome refers to a clinical syndrome in which the spinal cord is damaged due to external compression and internal spinal cord lesions, resulting in paralysis of the upper motor neurons of the ipsilateral limb below the level of the lesion, loss of deep sensation, impairment of fine touch, vasodilation and contraction, loss of pain and warmth in the contralateral limb, and preservation of bilateral tactile sensation, mainly in the cervical spine. . The resulting motor disorder can affect the patient’s walking, and the sensory disorder makes the patient prone to injury, especially the skin sensory disorder can lead to skin burns and other injuries, which can seriously affect daily life and cause the patient’s disability. Central canal syndrome: mainly occurs in cervical hyperextension injury. The cervical spinal canal undergoes dramatic volume changes due to cervical hyperextension, and the spinal cord is subjected to anterior and posterior compression by the folds of the ligamentum flavum, intervertebral discs or bone spurs, causing damage to the conduction bundles around the central canal of the spinal cord, resulting in paralysis of the extremities below the plane of injury, with the upper extremities heavier than the lower extremities, no sensory separation, and a poor prognosis. This injury is more common. The injury is mostly caused by cervical hyperextension injury; it can also be caused by obstruction of the root artery and anterior spinal artery during cervical spine injury, resulting in ischemia in the anterior, lateral and posterior columns of the gray matter of the spinal cord. The clinical features are that the severity of paralysis of the upper and lower extremities varies, with the upper extremity being heavier than the lower extremity, or one upper extremity may be paralyzed, or both lower extremities may be paralyzed without paralysis, with the upper extremity being the dominant area of segments 2-3 as a manifestation of lower motor neuron injury, and the lower extremity being a manifestation of upper motor neuron injury. Hand dysfunction is more obvious, and in severe cases, there is intrinsic hand muscle atrophy, and recovery is difficult. Tactile and deep sensory deficits may occur below the injured segment, and sometimes there is loss of sphincter function