Embolization is injected into the blood supply artery of uterine fibroids to block its blood supply artery, causing ischemia, degeneration, necrosis and resorption of the fibroids. Uterine fibroids are sex hormone-dependent tumors, and estrogen and pregnancy can promote the growth of fibroids. Arterial embolization prevents estrogen and progesterone from entering the fibroids, which creates a local hormonal environment similar to menopause and further shrinks the fibroids. The blood supply of uterine fibroids originates from the femoral arteries bilaterally without other anastomosing branches, and the branches form a rich vascular network within the pseudoenvelope around the fibroids, and enter the interior of the fibroids through radial branches, thickening, increasing, distorting and disorganizing its vascular network. The above characteristics of blood supply are the basis of arterial embolism. As there are extensive vascular anastomoses between bilateral femoral arteries and ovarian arteries, superior vaginal arteries, etc., the uterus will have blood supply without necrosis and maintain normal function as long as the fine branches and capillaries are kept open.