Facial muscle spasm (commonly known as facial twitching) is a paroxysmal involuntary twitching of half of the facial muscles. Typically, facial spasms begin on one side of the orbicularis oculi muscle and often begin as a paroxysmal twitching of the eyelid. Many people have experienced “eyelid twitching”. The eyelid twitching in facial spasm does not go away on its own, but gets progressively worse and more widespread over time, mostly spreading from the initial lower eyelid to all the muscles around the eye and then to the cheek. The rapid twitching (several times per second) of the periocular and cheek muscles during the attack causes muscle spasms that narrow the eye fissure and tilt the corners of the mouth to the side of the disease for a few seconds to tens of seconds, then resolves on its own, and in severe cases the broad neck muscles also twitch at the same time. The spasms can recur within a short period of time or stop for a longer period of time. Excitement, emotional stress, and conscious facial movement may induce spasms. The disease progresses slowly and does not usually resolve spontaneously. The age of onset peaks between 40 and 70 years of age. Prolonged spasms can result in a decrease in muscle strength of the affected expression muscles and a mild facial paralysis. Spasms of the orbicularis oculi muscle can lead to closure of one eyelid, preventing daily activities such as reading and driving a car. Frequent episodes of spasticity cause patients severe psychological stress and fear of attending social events and appearing in public, affecting their normal life. Etiology: A variety of causes can lead to facial spasm, such as: vascular compression of the facial nerve, recovery from facial neuritis (Bell’s palsy), occupying lesions of the pontocerebellar horn such as tumors and aneurysms, vascular malformations of the cerebellum, and facial hyperactivity. Among them, vascular compression is the most common factor. Treatment I. Surgical treatment: Microvascular decompression is the only treatment method that can cure facial myasthenia gravis. Surgical method: Patients need to be hospitalized for surgical treatment, and the average hospital stay is 10 days. Under general anesthesia, an incision of about 4 cm is made behind the affected ear, and a skull window of about 2 cm is opened, and the blood vessels compressing the facial nerve are cushioned with a special material under the operating microscope. The whole operation is completed within 1 hour with almost no damage to the patient’s nerve and brain tissue. The healing rate is over 98%. Complications are less than 1%. Second, other treatment 1, drugs: poor efficacy. Carbamazepine, phenytoin, baclofen and various sedative and tranquilizing drugs can reduce the symptoms for a few patients. 2, Chinese medicine: Chinese medicine, acupuncture, etc. The effect is not good. 3, botulinum toxin injection method: early complete relief up to 80%-100%, but generally 12-16 weeks, the symptoms recur after botulinum toxin metabolism, must repeat the injection. Common complications include: facial paralysis, dry eyes, diplopia, swallowing difficulties, etc. After 3 years of cumulative injections, complications amount to 60-75%.