Patients with hepatogenic diabetes mellitus have improved blood glucose and cured diabetes mellitus as liver function improves or liver disease is cured. Hepatogenic diabetes is often characterized by elevated postprandial blood glucose and normal or low preprandial blood glucose due to impaired sugar utilization caused by damage to the patient’s liver function, which causes impaired synthesis of hepatic glycogen. With the improvement of liver function or the cure of liver disease, the utilization of sugar by liver cells is restored and the synthesis of liver glycogen is restored, so that the patient’s postprandial blood glucose is normal. Therefore, the prognosis of blood glucose for patients with hepatogenic diabetes depends on the recovery of liver disease, and if liver function damage is restored and liver disease is cured, the patient’s blood glucose can also be restored.