Congenital heart disease is one of the common congenital malformations in children, which is caused by abnormal development of the heart and blood vessels during the fetal period, and the child is born with cardiovascular vascular lesions. The clinical manifestations of congenital heart disease are closely related to the pathological anatomy and pathophysiological changes caused by the congenital malformation. Some congenital malformations, such as simple bilateral superior vena cava, do not cause significant anatomic and pathophysiologic changes, and the patient may have neither symptoms nor signs; some congenital malformations, such as simple right-sided heart, do not cause significant pathophysiologic changes, and the patient has no symptoms, but the anatomic changes of the heart shifting to the right thoracic cavity lead to specific signs. Most of the precordial diseases have certain clinical symptoms and signs, and the symptoms appear early and obvious in patients with right-to-left shunt type, while patients with no shunt type and left-to-right shunt type have mild symptoms and appear late if the lesions are mild, but those with more severe lesions may have symptoms earlier; the most obvious sign is the typical murmur. Domestic Huang Kewen reported that the main clinical manifestations of neonatal precocious heart disease are characterized by heart murmurs, cyanosis, shortness of breath, heart failure, feeding difficulties and weight gain. 1, symptoms: The symptoms of precardiac disease are closely related to the type of disease, the hemodynamic changes of the lesion and its severity. Common symptoms include palpitations, shortness of breath, cough, hemoptysis, chest pain, easy fatigue, headache, dizziness, syncope, cyanosis, squat distance and swelling, etc. Infant patients also have difficulty swallowing, poor feeding, weight loss, vomiting, easy sweating and susceptibility to respiratory infections. The respiratory symptoms are related to pulmonary congestion, pulmonary ischemia, reduced oxygen content, tracheal compression or heart failure; chest pain, easy fatigue and central nervous symptoms are related to insufficient blood and oxygen supply to the coronary arteries, the whole body and the brain; cyanosis and squatting are common in patients with right-to-left shunts, which are caused by low arterial oxygen saturation and systemic hypoxia; edema and oliguria are often seen in congestive heart failure; Digestive system symptoms are mainly due to esophageal compression and stasis of the digestive system caused by congestive heart failure; the enlarged heart or large blood vessels compressing other organs (such as the recurrent laryngeal nerve) may also cause corresponding symptoms (such as hoarseness, etc.). In addition, the common complications of precordial disease are: infective endocarditis, pneumonia, heart failure, arrhythmia, brain abscess, thromboembolism, etc. 2, signs: most of the precordial diseases have characteristic heart or vascular murmurs, abnormal heart sounds, most of these murmurs are accompanied by tremors, the nature, the main auscultation site, distribution and conduction direction vary with the different malformations. Other common signs include dysplasia, cyanosis, pestle-like fingers (toes), thoracic deformity, enlargement of the heart’s turbinate, elevated pulsations in the precordial region, and changes in blood pressure and pulse rate. Patients with precordial disease have poor systemic blood supply, so most of them are stunted, but milder lesions may have no effect on development, and some patients (such as aortic constriction) are taller; cyanosis and pestle-like fingers (toes) are seen in patients with right-to-left shunts; enlarged heart is the main cause of thoracic deformity in patients with this disease, and thoracic deformity is mainly anterior cardiac bulge, but also posterior or lateral spinal protrusion deformity; aortic The blood pressure of the upper extremity is often increased when the aorta is narrowed, reduced when the aortic orifice is severely stenosed, and the pulse pressure is widened when the arteriovenous catheter is not closed or the aortic valve is not closed, and causes corresponding changes in pulse palpation.