Abdominal tenderness is due to mild irritation or chronic inflammation of the peritoneum and is seen in all types of tuberculous peritonitis, but is generally considered to be a clinical feature of the adherent type of tuberculous peritonitis. The vast majority of patients have varying degrees of pressure pain, generally mild, with a few having significant pressure pain and rebound pain, the latter mostly seen in the caseous form. The majority of tuberculous peritonitis is secondary to tuberculous lesions in other organs. The route of infection can be either by direct spread of intra-abdominal tuberculosis or by hematogenous dissemination. The former is more common, such as intestinal tuberculosis, mesenteric lymphatic tuberculosis, and tuberculosis of the fallopian tubes, all of which can be direct primary foci of the disease. It is more common in women than men and may be due to retrograde infection of pelvic tuberculosis. 1, drug therapy is still based on the principle of adequate and combined treatment. The course of treatment should be at least 18 months. 2.For patients with ascites, after the release of ascites, intraperitoneal injection of dexamethasone acetate and other drugs can accelerate the absorption of ascites and reduce adhesions. 3.For patients with hematogenous dissemination or severe tuberculosis toxemia, adrenal glucocorticoids can be added on top of effective anti-tuberculosis drug therapy, but they should not be applied for a long time. 4.Most patients may have already received anti-tuberculosis drug treatment. Therefore, these patients should choose drugs that have not been used or used sparingly in the past and develop a combination drug regimen. 5.Surgical treatment is feasible in case of complicated intestinal obstruction, intestinal fistula and septic peritonitis. If it is difficult to differentiate from intra-abdominal tumor, abdominal dissection is feasible.