Shock and DIC can be causative and interact with each other, thus forming a vicious circle that can worsen the patient’s condition, endanger the patient’s life, and even cause the patient’s death. In the process of shock-induced DIC, the patient’s prolonged tissue cell ischemia, hypoxia, acidosis, and endotoxin combine to cause damage to the vascular endothelium, which in turn promotes the coagulation system and leads to widespread microthrombosis, thereby aggravating the further progression of shock. In the process of DIC-induced shock, the cardiac blood return is often insufficient due to extensive microthrombosis, and the effective circulating blood volume decreases, leading to further aggravation of shock. In addition, the decrease in blood volume due to extensive hemorrhage and the increase in intracardiac hemorrhage, which in turn leads to an increase in shock. Due to vasodilation, the activation of the associated complement system in the body can further cause vasodilatory dysfunction, resulting in microvascular dilation and increased permeability, which in turn can cause a further decrease in effective circulating blood volume and blood volume, which in turn can aggravate shock. Therefore, shock and DIC can be the cause and effect of each other, forming a vicious circle together.