1, neonatal type: about 12% of mothers with myasthenia gravis can transmit AchR-Ab (anti-acetylcholine receptor antibody) to their newborn infants through the placenta and cause myasthenia gravis. The child is born with a low cry, difficulty in sucking, low muscle tone, reduced movements, and especially the typical symptoms of respiratory insufficiency. After treatment, they are mostly cured within 13 months. 2, congenital myasthenia gravis syndrome: an inherited neuromuscular junction transmission disorder with a unique pathogenesis, less common, with severe symptoms and persistent extraocular muscle paralysis shortly after birth, often with a positive family history, but whose mother did not suffer from myasthenia gravis. 3. Juvenile type: Pathophysiologically and symptomatically similar to the adult type. The onset of the disease is mostly after the age of 10 years, with simple extraocular muscle paralysis, some with swallowing difficulties and limb weakness. Juvenile patients with myasthenia gravis account for 11%-29% of all patients, with an annual incidence of 1.1/1 million. Clinical manifestations In children, the extraocular muscles are most commonly involved, causing ptosis, diplopia, and ocular fixation, which is present in up to 90% of children. Diplopia may not be initially dominant, but can be triggered by sustained vertical gaze. Prognosis The prognosis is good in children with a benign course. A small percentage of pediatric patients have spontaneous remission, and approximately 75% of pediatric patients have remission after a combination of Western and Chinese medications.