The rate of prenatal ultrasound diagnosis of fetal esophageal atresia is not high, at about 60%, depending largely on the knowledge of the ultrasonographer of the atresia. Usually there are five types of esophageal atresia, the most common being type III, which accounts for about 85%, followed by type I, which accounts for about 2%, and other types are rare. The application of fetal MR examination on this basis can increase the diagnostic rate to 80%, so MR examination is routinely required in fetuses suspected of having esophageal atresia on ultrasonography. The main points of ultrasonography diagnosis are: 1. the fetal gastric vesicles disappear or become smaller, especially under dynamic observation, the disappearance of gastric vesicles requires a high degree of vigilance; 2. excessive amniotic fluid, the amount of amniotic fluid in normal pregnancy increases with the increase of gestational weeks , where the amount of amniotic fluid exceeds 2000 ml in any period of pregnancy, it is called excessive amniotic fluid. Excessive amniotic fluid from esophageal atresia can lead to preterm delivery. It has been described that a dilated blind pouch (PONCH SJGN) can be seen at the proximal end of the fetal esophagus under dynamic observation, but by our observation, it has not been seen in confirmed cases of fetal esophageal atresia. The diagnostic points of the examination of MR: 1. There is a clear diagnostic basis for ultrasonography; 2. The blind end of the dilated esophagus can be clearly seen in the fetal anterior thoracic region. 3.It can be determined that the fetus has excessive amniotic fluid, so that the fetus is in the package of large amount of amniotic fluid. This is particularly evident in the type I pathology. 4. The absence of a normal gastric blister shadow in the region of the fetal stomach can be seen. Those with confirmed or suspected fetal esophageal atresia that clearly require delivery need to be treated promptly before or during labor and delivery and postpartum. The specific treatment measures are: 1. Excessive prenatal fetal amniotic fluid is an important factor leading to preterm delivery of fetuses with esophageal atresia, so timely treatment is needed when there is too much amniotic fluid. The method of intermittent amniotic fluid release can be adopted, and each release of amniotic fluid should not exceed 1500 ML, so that the fetus can be delivered at full term or weigh more than 2 KG at birth as far as possible. 2. Choose to deliver in a medical center with neonatal resuscitation. As fetuses with esophageal atresia are often born with amniotic fluid aspiration, so as far as possible, make good perinatal management and respiratory care at birth to reduce or avoid the occurrence of neonatal pneumonia or amniotic fluid aspiration syndrome. 3.If you cannot feed after birth, you need to feed only after the diagnosis is clear and esophageal atresia is excluded. 4. Confirmation of the diagnosis after birth needs to be made within three days, and a simple method is difficulty in inserting a gastric tube or the phenomenon of gastric tube regurgitation. Imaging is the main method to confirm the diagnosis and can clarify the type of pathology, providing a reliable basis for treatment. 5.Surgical treatment needs to be performed within three days to minimize neonatal pneumonia produced by reflux. 6.For long segmental atresia with a distance greater than 3.5CM, we are currently taking a new approach: using a free gastric coiled tube with a tip to anastomose the substituted esophagus in parallel, which has achieved very satisfactory results, with the advantage that one phase of treatment is completed, and the second is that the situation of postoperative esophageal stricture and complications of esophageal reflux is significantly improved, which is currently the best choice for the treatment of long segmental esophageal atresia. Combining prenatal diagnosis and treatment of fetal esophageal atresia with postnatal treatment in the neonatal period has greatly improved the cure rate of esophageal atresia, and our current cure rate has reached 90%, which is close to the international European and American levels.