Central nervous system disorders such as stroke, traumatic brain injury and encephalitis, after a period of onset, abnormal muscle excitability such as limb stiffness and spasticity occurs, which mostly affects the patient’s motor function. Among them, the most common one, which occurs in the lower limbs and hinders the walking function the most, is called clubfoot. It is named horseshoe because it resembles the appearance of a horse’s hoof drooping when the toe is raised. The so-called inversion refers to the phenomenon that when the foot swings or lands, the inside of the foot is high and the outside is low and cannot be flattened. As the foot and ankle turn inward and downward, the floor or wheelchair tread will rub against the lateral edge of the foot, which can seriously damage the skin. When the affected foot is supporting the weight, the inversion may increase, causing instability of the ankle joint and affecting the lower limb force and body balance; when the affected foot is supporting, it is difficult to dorsiflex the ankle joint upward, which will prevent the other side of the lower leg from crossing to the front of the supporting foot; when the affected limb is swinging forward, the toe rubbing the ground will affect the foot leaving the ground and body movement, and the propulsion force of the ankle joint will be significantly reduced. The propulsive force of the ankle joint is also significantly reduced. In addition, clubfoot can also indirectly cause abnormal knee and hip movements. The abnormal excitable muscles involved in clubfoot can vary from patient to patient. The clubfoot is mainly caused by abnormal contraction of the gastrocnemius and the hallux valgus complex, but in individual cases the abnormalities of the two muscles are not consistent. Other teratogenic forces in horseshoe foot can come from abnormal contractions of the long toe flexors and the internal foot muscles, when the patient may experience toe curling and toe pain. Ankle and foot pronation is caused by an abnormality in one or more of the posterior tibial, anterior tibial, or longissimus bunions muscles. When the long bunion extensor muscle goes into spasm, the patient will have a buckling of the big toe and can experience pain at the tip of the bunion and pain under the first metatarsal head when the affected foot is weighted. The clubfoot can also change: in the early stages, the muscle abnormality may reverse; in the later stages, the muscle tendon may undergo contracture degeneration and the deformity may become increasingly fixed. If the central nervous system is completely normalized before the muscle degeneration, clubfoot can disappear. Unfortunately, however, the vast majority of central disorders that cause clubfoot are unlikely to recover completely. Therefore, treatment of the clubfoot itself is a practical rehabilitation strategy that can improve walking. If the patient has generalized muscle spasm, of which clubfoot is only a part, then the patient will need oral medications such as baclofen and tizanidine, or if the spasm is too severe, intrathecal implantation of a baclofen pump or dorsal rhizotomy of the spinal nerve may be considered. If the clubfoot is a more limited lesion, treatments such as retraction, orthotics, antagonistic muscle electrical stimulation, botulinum toxin injections, and orthopedic surgery can be given depending on whether the lesion is reversible or not. Among them, botulinum toxin local target muscle injection is one of the main treatment modalities for focal clubfoot, which has been widely used in developed countries such as Europe, Japan and the United States, and some hospitals in China have also mastered this treatment technology. Other treatment modalities have been widely carried out in medical institutions at all levels in China. For example, residents of Shanghai who have obtained a disability certificate can receive a discount on the purchase of orthopedic devices through the relevant agencies of the Disabled Persons’ Federation.