Buga’s syndrome (in ferior vena cavasyndrome) is a series of clinical syndromes caused by partial or complete obstruction of the inferior vena cava due to invasion, compression or intracavernous thrombosis of the inferior vena cava. In the acute phase of simple hepatic vein thrombosis, patients have fever, right upper abdominal pain, rapid onset of massive ascites, jaundice, hepatomegaly, tenderness in the liver area, and oliguria. Death can occur within days or weeks due to circulatory collapse (shock), liver failure, or gastrointestinal bleeding. The non-acute phase of simple hepatic vein thrombosis is characterized by portal hypertension, hepatosplenomegaly, intractable ascites, and bleeding from ruptured esophageal varices. In simple inferior vena cava obstruction, there are superficial varices in the thoracoabdominal wall and back (venous blood flow from bottom to top) and varicose veins, swelling, hyperpigmentation and ulcers in the lower extremities. Patients may have shortness of breath due to obstruction of the hepatic veins and inferior vena cava and reduced blood return to the heart. It varies according to the number of vessels involved, the degree of involvement and the nature and status of the obstructing lesion. It can be divided into acute, subacute and chronic forms. In advanced stages, patients may have a typical “spider” physique due to malnutrition, protein loss, increased ascites, and wasting. Treatment includes interventional therapy, medical treatment and surgical treatment: 1. Interventional surgery: interventional surgery is preferred for Buga syndrome, which is less invasive and more effective. If the inferior vena cava or hepatic vein is combined with thrombosis, it can be treated with intubation thrombolysis first, and balloon dilation is feasible after complete dissolution of the thrombus to widen the narrowed section. If the effect of balloon dilation is poor, the hepatic vein and or inferior vena cava stenting treatment is feasible. Internal treatment includes low-salt diet, diuretic, nutritional support, autologous ascites transfusion or concentrated transfusion, etc. Patients in the acute phase with simple thrombosis within 1 week of onset can be treated with anticoagulants, but most cases are not diagnosed until weeks or months after thrombosis. In most cases, conservative treatment may buy time for collateral circulation to develop, but the patient will eventually require surgery. Patients with Bard-Gialli syndrome, especially in advanced stages, often have intractable ascites and severe malnutrition. As a supportive therapy before surgery, medical treatment can improve the patient’s general condition, reduce surgical mortality, and facilitate the patient’s postoperative recovery. 3, surgical treatment (1) septal laceration: septal laceration via the right atrium: the method is to enter the thoracic cavity through the right anterior 4th rib external thoracic incision or through the sternal incision, and cut the pericardium longitudinally in front of the right phrenic nerve. (2) Inferior vena cava-right atrial shunt: (3) Superior mesenteric vein-right atrial shunt, intestinal cavity shunt, intestinal cavity-atrial shunt, and intestinal neck shunt, which are variously invasive and have received positive clinical evaluations. (4) Radical surgery: For septal type cases with high location of vena cava obstruction, the lesion can be removed by dissecting the thoracic segment and part of the ventral segment of the inferior vena cava after dissecting out the thoracic segment and controlling both ends of the lesion. If the obstructive lesion is extensive or there is a large amount of thrombosis distally, the inferior vena cava of the hepatic segment can be dissected longitudinally under extracorporeal circulation, the septum and thrombus can be removed, the hepatic vein can be explored and its patency restored, and the inferior vena cava can be repaired with Gore-Tex or Dacron patches. Although radical surgery directly removes the primary lesion, there is still a possibility of recurrence in cases with concomitant inflammation of the inferior vena cava. Depending on the condition, surgical interventions such as portal vein dissection, splenectomy, and splenorenal vein shunt are performed to reduce portal vein pressure and cure hypersplenism. Surgery requires surgery on large blood vessels, which is not only traumatic and slow to recover, but also complex and risky. It should be seen and treated in a hospital with experience and basic strength.