AVM is a type of intracranial vascular malformation, a congenital disease in which the size and shape of the lesion grows and changes with the patient’s age, and is the main cause of intracerebral hemorrhage in young people. AVMs are relatively uncommon in hemorrhagic cerebrovascular diseases represented by intracranial aneurysms. More than half of patients present with ruptured hemorrhage, each bleeding event carries a 30% risk of permanent disability, 15% risk of death, and is the most important risk factor for rebleeding. Second, approximately 25% of patients present with epilepsy, 15% with headache, and 5% with focal neurological symptoms such as speech or limb movement disorders. At present, craniotomy, intracranial interventional embolization and stereotactic radiotherapy are the three basic means of treatment for cerebral AVM. since 2004, the Interventional Department of Henan Provincial People’s Hospital has treated more than 100 cases of symptomatic cerebral arteriovenous malformation with intracranial embolization, from the initial surgical silk segments, PVA particles, IBCA colloid to the current internationally popular Onyx embolization materials, from simple embolization of the blood supply artery, non selective embolization of malformed vessels, to selective target embolization, from pure reliance on embolization to combined application of multiple techniques, which reflects not only the progress of technical materials, but also our doctors’ in-depth understanding of the anatomy of malformed vessels, the disease itself and the advantages of endoluminal techniques. We believe that in symptomatic cerebral AVM, some factors are independent risk factors for hemorrhage from the lesion, such as aging, deep brain lesions, and previous hemorrhage from the lesion; certain features of the lesion itself also increase the risk of rupture and hemorrhage, including: the presence of a hemangioma within the lesion, the presence of an aneurysm on the blood supply artery, a single draining vein, a poorly draining vein, location within or adjacent to the ventricles, and small size. The above-mentioned cases tended to be treated with aggressive intervention. To retrospectively analyze and summarize the treatment experience of these cases, the origin, natural history, pathology, clinical and therapeutic regression of cerebral AVM as a complex neurovascular lesion remains to be further investigated. We have already had the experience of a large number of cases in international practice, and we are intensifying systematic follow-up to assess the “long-term outcome of non-cranial treatment of symptomatic cerebral AVM” using the NIHSS and mRS scores. We believe that there is no simple AVM, and each AVM has its own unique vascular architecture and hemodynamic characteristics, so we will analyze its specific structure and hemodynamic characteristics based on DSA and MRI, and develop appropriate embolization strategies, combining spring coils, NBCA, Glubran, Onyx, etc. Multiple embolization techniques are possible to benefit most patients with symptomatic cerebral AVM.