Paroxysmal atrial tachycardia (PAT) is a sudden onset of a regular, rapid set of heart rates (frequency 160-200 beats/minute) triggered within the atria. What happens when paroxysmal atrial tachycardia occurs Most patients have a sudden onset of tachycardia that stops abruptly, lasts for seconds, minutes, hours, or days, and disappears on its own. During the attack, there are palpitations, precordial discomfort, nausea, vomiting, and occasionally polyuria; those with pre-existing heart disease may experience angina pectoris, fainting, decreased blood pressure, or heart failure. The heart rate during the attack is fast and uniform, 160-220 times per minute, with a tendency of repeated attacks and frequent attacks. What are the symptoms? There are several possible mechanisms leading to the development of PAT. PAT can be triggered by a single atrial premature beat, a rapid heart rate that tends to occur and end abruptly, and whose episodes can last from several minutes to several hours. In addition to the common palpitations, PAT is often accompanied by other symptoms, such as fatigue. Usually the patient’s heart is normal and not at great risk. What is the treatment? Short episodes that do not affect the hemodynamic force do not require special treatment, especially if they occur in the absence of organic heart disease. Treatment to stop the episodes can be started by trying various mechanical methods to stimulate the vagus nerve, such as ① exhaling forcefully after holding the breath. (2) Stimulation of the throat to cause nausea. ③ compression of one side of the eyeball (after closing the eye with the thumb compression of the eyeball) or carotid sinus (used to point to the direction of the cervical spine compression of the upper edge of the thyroid cartilage level carotid artery pulsation most obvious), first compression of one side 10-30s, if not effective then try the opposite side. It is not advisable to compress both sides at the same time, otherwise there is a risk of blocking the blood supply to the brain or causing cardiac arrest. The heart should be auscultated at the same time as compressions, and compressions should be stopped as soon as the tachycardia stops. If the above methods do not work, putting the patient to sleep may also end the tachycardia. Most people generally seek pharmacologic intervention. Isoptin or adenosine is often given intravenously to terminate the attack. Electrical cardioversion is used only when medications are ineffective, although this is quite rare. Prevention is more difficult than treatment, but there are several drugs that may be effective alone or in combination. In some patients, the presence of an abnormal conduction pathway requires treatment with radiofrequency ablation. What I can do Patients with tachycardia must pay attention to the improvement of their overall physical fitness and actively participate in qigong exercises. Be open-minded, optimistic and cheerful, and treat people with stability when they encounter difficulties, and do not be irritable and angry. If the tachycardia is not organic heart disease, and I have no obvious pain, do not have to rely on drugs. When the attack can stop heavy or stressful work, rest for a moment, you can recover. Avoid sexual intercourse to nourish Yin essence and suppress heart fire. Avoid eating hot and spicy food, such as ginger, chili, tobacco, curry, coffee, chocolate, strong tea, etc. The first food that nourishes Yin and blood and calms the mind, such as animal liver and kidney, turtle, turtle, black and white fungus, lotus, walnuts, cinnamon, honey, lily, mulberry fruit, etc. Do not watch intense and stimulating films and participate in strenuous and stressful recreational activities. The body’s biological clock should follow the natural rhythm. What may happen The atrial rate of 160-250 beats per minute is called paroxysmal atrial tachycardia. It is common in people without organic heart disease, but also in people with rheumatic heart disease, left atrioventricular valve lesions, coronary artery disease, hypertensive heart disease, hyperthyroidism, cardiomyopathy, and pre-excitation syndrome. It can also be caused by digitalis toxicity, which is often accompanied by atrioventricular block. Abnormally high autoregulation at atrial ectopic pacing sites; sinus node and atrial junctional sites and intra-atrial circular folding motion or triggered excitation.