Redness of the tympanic membrane and redness behind the ear are most often seen in patients with intracranial venous sinus occlusive cranial hypertension, most patients have perforated tympanic membrane with pus, and some patients have redness of the tympanic membrane and redness behind the ear. Intracranial venous sinus and venous thrombosis is the main cause of cerebral venous reflux and impaired cerebrospinal fluid absorption, which results in cranial hypertension, and this particular type of cranial hypertension is called venous sinus occlusive cranial hypertension. Transverse sinus thrombosis is mostly due to otitis media or mastoiditis lasting several weeks, the patient has headache, which is aggravated by head rotation, vomiting, skin vein congestion behind the ear, and if phlebitis has extended to the internal jugular vein, there is tenderness in the neck, optic disc edema, which is mostly confined to the diseased side and is not severe, and focal encephalitis symptoms are convulsions and light hemiparesis on the opposite side. When the left transverse sinus thrombosis can manifest aphasia, most patients have tympanic membrane perforation and pus flow, and some patients have red tympanic membrane and postauricular redness. The diagnosis of intracranial venous sinus occlusion mostly depends on clinical manifestations, such as headache, nausea, vomiting and optic disc edema of cranial hypertension, as well as local neurological localization signs and local infection lesions related to venous sinus thrombosis and related systemic factors. In recent years, due to the extensive and widespread use of antibiotics, venous sinus thrombosis caused by local infection tends to have a subacute and chronic onset, and some patients have only chronic cranial hypertension and lack other typical manifestations of venous sinus thrombosis, showing only symptoms of high cranial pressure. Differentiation is due to tympanic membrane congestion due to various ear inflammations or ear trauma, and tympanic membrane rupture. Postauricular mastoid edema is a clinical symptom of acute purulent mastoiditis. The mastoid mucosa is continuous between the mastoids of the middle ear, and the mastoid mucosa has a corresponding inflammatory reaction after a purulent infection of the middle ear, which is initially cicatricial and may have mild pressure pain in the mastoid area, and the mastoid inflammatory reaction disappears as soon as the tympanic membrane is perforated and pus flows. If the pus is not drained smoothly, or if type III pneumococcal infection, which is very toxic, destroys the bone and causes edema, hemorrhage, necrosis and suppuration of the mucosa of the mastoid process, it becomes acute purulent mastoiditis, which is actually a follow-up to acute otitis media and should be called acute purulent mastoiditis of the middle ear. The examination of purulent auricular chondromyelitis reveals redness and swelling of the auricle. Auricular perichondritis can be divided into two types: plasmacytic and purulent. The lesions are either serous exudate (plasmacytic) or pus formation (purulent) between the cartilage and the perichondrium. Prevention is aimed at the causes of venous sinus occlusive cranial hypertension, treatment of chronic otitis media and some systemic diseases that may cause the disease, such as extracranial tumors, blood disorders, and metabolic disorders.