Arteriovenous malformations are abnormalities of the vessels that make up the connection between the intracranial arterial and venous systems and the lack of a capillary bed between them. Approximately 2% of the lesions are multiple and the rest are solitary. The prevalence is comparable in males and females. Arteriovenous malformations are the leading cause of nontraumatic intracranial hemorrhage in adolescents, and they are the leading cause of neurological deficits or death in patients younger than 20 years of age. 1. Clinical manifestations: Arteriovenous malformations can manifest as seizures, headaches, focal neurological dysfunction or hemorrhage. Intracranial hemorrhage is the most catastrophic and frightening. 2.Examinations: CT: The bleeding of arteriovenous malformation can be found, and the general extent of vascular malformation, blood supply arteries and drainage veins can be seen by enhancement. MRI: low-signal, distorted blood supply arteries, lesions and draining veins can be seen in the area of vascular malformation. Cerebral angiography (DSA): three basic features are demonstrated: the blood supply artery, the lesion and the draining veins. A very important but not specific angiographic sign is the visualization of the draining veins in the arterial phase, a feature that identifies an arteriovenous short circuit. 3. Treatment: Microsurgery is the gold standard for definitive treatment of arteriovenous malformations. Indications for surgical treatment are classified according to clinical symptoms: epilepsy, headache, focal neurological dysfunction, or hemorrhage. Appropriate use of antiepileptic drugs to control seizures and surgery may convert patients with arteriovenous malformations from refractory epilepsy to drug control. Patients presenting with seizures after resection of the action vein malformation alone (without cortical resection) are cured by surgery in 56% of patients, with the remainder having persistent epilepsy. Epilepsy is cured in about 75% after resection of the action vein malformation plus cortical resection. Both of these surgical treatments have superior outcomes to embolization or ligation treatments, and if there is a high suspicion that the patient’s headache is related to the arteriovenous malformation, the clinical outcome of surgical treatment may be excellent. Stereotactic radiosurgery has been shown to be effective in treating these conditions. Approximately 80% of arteriovenous malformations with lesions less than 3 cm can be occluded after treatment, with the time from treatment to occlusion varying from 2 to 3 years, during which the patient is not guaranteed not to bleed because of the extended time between radiation treatment and completion of occlusion. Embolization is another therapeutically effective treatment, but if the arteriovenous malformation has more than three supplying arteries, the chances of complete occlusion by endovascular treatment alone are almost non-existent. Major embolization may be performed followed by reduction of the lesion and then radiation therapy.