Tinea Auricularis starts as a red papule and blister. It then flakes off and gradually spreads and expands in all directions into a large erythematous plaque covered with scales. The edges are clear and there are papules or blisters. It may extend to the external auditory canal, the whole ear, and the neck and face. Patients often have a history of topical corticosteroid use. Self-perceived pruritus is evident, often on one side. There are many causes of auricular fungus, see the following description of the causes of auricular fungus: Otomycosis is a common disease, accounting for 15% to 20% of ear infections. It occurs mostly in warm, humid tropical and subtropical areas. It can occur at any age, but is most common between the ages of 20 and 40. It is common unilaterally and more often on the right side, probably related to the convenience of digging the ear with the right hand. It is more common in summer. The main fungi that cause auricular infections are dermatophytes such as Trichophyton rubrum, Trichophyton rubrum and woolly microsporum. It originates from direct spread of ringworm on the head and face or direct or indirect contact transmission of ringworm on the hands and feet, body and nails, etc. This type of infection is also called tinea capitis. The vast majority of pathogens that cause infections of the external ear canal are Aspergillus fumigatus and Aspergillus niger. Aspergillus niger accounts for more than 90% of them. Other Candida, Ken mold, plow mold, column top spores, short broom mold, root mold, common head mold, etc., more from the drift of spores in the air. 80% to 90% of otitis externa can be found in a variety of bacteria, such as Pseudomonas, Aspergillus, micrococcus, Streptococcus, Escherichia coli and rod-shaped bacilli, etc., when coexisting with fungi. It can aggravate the fungal infection.