Ball palsy is medulla oblongata paralysis. Because the medulla oblongata is also called the medullary bulb, the medullary palsy is referred to as bulbar palsy, also known as true bulbar palsy. When the motor nerve nuclei within the medulla oblongata, or the cranial nerves from the medulla oblongata (including the glossopharyngeal nerve, vagus nerve and hypoglossal nerve), are paralyzed due to disease, a group of symptom clusters will appear. The main manifestations are choking on drinking and eating, difficulty in swallowing, hoarseness or loss of voice, etc. Therefore, any lesion that directly damages the medulla oblongata or related cranial nerves is called true bulbar palsy. If the lesion is in the pontocerebrum or above, causing loss of innervation of the motor nucleus in the medulla oblongata, the medulla oblongata palsy is called pseudobulbar palsy. In other words, true bulbar palsy damages lower motor neurons, while pseudobulbar palsy damages upper motor neurons; and the lesion site of true bulbar palsy is mostly unilateral suspensory nucleus, while the lesion site of pseudobulbar palsy is bilateral cortical or cortical medullary bundle; in terms of medical history, true bulbar palsy is mostly the first onset, while pseudobulbar palsy is multiple onset; the former has no strong crying and laughing performance, while the latter has; the former has tongue muscle fibrillation and atrophy, while the latter has The former has lingual muscle fibrillation and atrophy, while the latter has no such symptoms; the former has no gag reflex and brainstem reflex, while the latter has them; the former has no cone fasciculations, while the latter has them; the former has no urinary disturbance, while the latter has them; in addition, the EEGs of the two are also very different, with the former having no abnormalities, while the latter has diffuse abnormalities.