Etiology Congenital aortic constriction manifests as a restrictive narrowing or atresia of the aorta, often near the opening of the original ductus arteriosus in the aortic isthmus; acquired aortic stenosis is mainly due to aortitis, atherosclerosis, and aortic dissection. Only stenoses located in the aortic arch, descending aorta, and superior abdominal aorta cause clinically significant hypertension. The basic pathophysiological changes of the disease are redistribution of blood flow due to stenosis and ischemia of renal tissue causing water and sodium retention and RAS activation. Diagnosis Hypertension in the upper extremities, weak or absent pulses in the lower extremities, significantly lower blood pressure in both lower extremities than in the upper extremities (ABI < O.9), and a significant vascular murmur around the stenotic vessels on auscultation. Non-invasive imaging tests such as Doppler ultrasound, magnetic resonance angiography, and computed tomography angiography can clarify the site and degree of stenosis. Treatment Once aortic stenosis is diagnosed, with diameter stenosis ≥ 5% and systolic pressure difference between distal and proximal ends of the lesion ≥ 2O mmHg, if there is no contraindication to surgery, early surgery should be performed to remove the stenosis and achieve the goal of radical treatment. Surgical methods include surgical treatment and interventional treatment. If the stenosis is limited and there are no important collateral vessels, interventional treatment is preferred.