For mitral regurgitation mild do not worry too much if there is no other discomfort, generally do not need treatment, to understand the mitral and tricuspid regurgitation, should start with the structure and function of the valve. Mitral valve, tricuspid valve is the human heart between the left atrium, left ventricle and right atrium, right ventricle between the two automated live valve door, when the heart diastolic, valve live door open, blood flow from the atrium to the ventricle. When the ventricle fills and then contracts, the valve valves close tightly, and the blood that is strongly pumped from the ventricle enters the large and small circulation (also called the body and lung circulation) through the large vessels. Theoretically, the closure of the valve valve should be seamless and there should be no blood flow back to the atria when the ventricle is closed. However, due to continuous advances in ultrasound instrumentation, it has been found that regurgitation of the mitral and tricuspid valves, especially minor regurgitation, is quite common. The cause of regurgitation can be organic or functional (or physiological). However, it is generally accepted that mild mitral regurgitation does not need to be treated if the other tests are clear. A variety of organic heart diseases can impair the opening and closing function of the valve, resulting in a condition in which the valve does not open (valve stenosis) or does not close tightly (incomplete closure of the valve, resulting in blood regurgitation). In our country, the common cause of mitral valve closure insufficiency is rheumatic heart disease, and others such as congenital malformation of the valve and calcification of the mitral annulus. It should be noted that any heart disease causing left ventricular enlargement may lead to mitral valve insufficiency, such as coronary artery disease, hypertrophic cardiomyopathy, hypertensive heart disease, mitral valve prolapse, etc. The most common cause of tricuspid valve insufficiency is mostly functional. Therefore, patients with mitral regurgitation and tricuspid regurgitation should first exclude heart disease. For mild mitral regurgitation, a comprehensive analysis of clinical data should be performed, including medical history, symptoms and relevant laboratory tests, presence of cardiac enlargement, heart murmur, etc. The ultrasound data you provided shows that the size of the atrial internal diameter, valve morphology and opening and closing motion are normal, and no evidence of organic heart disease has been detected, so when the findings suggest only minor regurgitation of the mitral and tricuspid valves, functional regurgitation should be considered. If necessary, a local specialist can be consulted for further diagnosis and regular clinical follow-up. Asymptomatic or mildly symptomatic patients do not require treatment and can work and live normally with regular follow-up. Those with a history of syncope, family history of sudden death, complex ventricular arrhythmias, or Marfan syndrome should avoid excessive physical work and strenuous exercise. In cases of chest pain, receptor blockers can be used to reduce myocardial oxygen consumption and ventricular wall tension, slow down the heart rate, weaken myocardial contractility, and improve the degree of mitral valve prolapse, thus relieving chest pain. Nitrates can aggravate mitral valve prolapse and should be used with caution. For those with mitral valve insufficiency, antibiotics should be applied prophylactically before and after surgery, tooth extraction, childbirth, or invasive examinations to prevent infective endocarditis. For arrhythmias with a history of palpitations, dizziness, vertigo or syncope, receptor blockers may be used, and when ineffective, phenytoin sodium, quinidine, etc., may be used in combination if necessary.