What is intracranial injury and will there be any after-effects?

  Injuries caused by violence to the skull. These include soft tissue injuries to the head, skull fractures and brain injuries. Among them, brain injury has serious consequences and should be especially vigilant. The cause of the disease is common in accidental traffic accidents, industrial injuries or firearm operations.  Soft tissue injuries in the subscalp hematoma more, without special treatment, often can be self-healing. Scalp lacerations bleed a lot and should be cleared and sutured early. Thread-like fractures of the skull cap do not require treatment. Larger depressed fractures should be repaired early. Fractures at the base of the skull often cause cerebrospinal fluid nasal leakage or ear leakage should be considered as open cranial injury, which is highly susceptible to retrograde infection, so the treatment of cerebrospinal fluid leakage is drainage without blockage, anti-inflammatory to be self-healing, and a few non-healing cases can be elective surgical repair.  Primary brain injury is commonly concussion, the patient has a positive history of trauma, loss of consciousness immediately after the injury, a short period of time awake, often can not recall the instantaneous process of injury, symptomatic treatment can be healed, brain contusion and contusion is the top of the occipital landing to form a hedge injury, brain tissue in the cranial block movement, and the anterior cranial concavity and middle cranial concave bottom friction, resulting in brain tissue contusion or contusion, can cause traumatic subarachnoid hemorrhage (headache, nausea , vomiting, neck resistance, and lumbar puncture may have bloody cerebrospinal fluid), which should generally be closely observed for timely detection of intracranial hematoma.  Secondary brain injury commonly includes cerebral edema and intracranial hematoma. On the basis of brain injury, vasogenic cerebral edema is formed, which can be local or whole-brain; if the contusion is heavy and local bleeding is more frequent, subdural hematoma or intracerebral hematoma can be formed. If the temporal skull fracture damages the middle dural artery, an epidural hematoma may be formed. All the above pathological changes can be secondary to increased intracranial pressure and even form brain herniation, which can endanger life.  Cerebral edema should be treated conservatively (e.g., dehydration, administration of hormones and restriction of intake). Intracranial hematoma is in principle an open hematoma removal, and should be operated early, once the formation of brain herniation, the prognosis is dangerous.  Does craniocerebral injury have any effect on pediatric intelligence?  Craniocerebral injury is more common in pediatric patients. Even if the injury is not serious from the outside, it may still cause damage to the brain tissue and affect the development of intelligence.  Craniocerebral injuries are caused by falls from heights, window falls, stairway slips and falls, pediatric fights, or injuries due to traffic accidents with bicycles and motor vehicles. Children with cranio-cerebral injuries should be promptly diagnosed and treated in the hospital. Wounds should be cleared and sutured to prevent infection, tetanus antitoxin injection. And for non-wound cranio-cerebral injury should be closely observed, pay attention to rest, so as not to aggravate the brain contusion or concussion. If the headache worsens, vomiting and coma appear, a doctor should be consulted again. In some cases, even within 2 to 3 weeks after the injury, due to the increase in intracranial hematoma, there are convulsions, coma, vomiting and other manifestations of increased intracranial pressure. According to statistics, about 1/4 of children with craniocerebral injury may have decreased intelligence within 1 year after the injury, manifested as indifferent expression, mobility, reduced control, lack of mental concentration, poor memory, etc. A small number of patients still have sequelae such as headache, hemiparesis, mental retardation, and epilepsy 1 year later.