Gynecologic infections are common disorders in women and are caused by various pathogenic bacteria and microbial infections of the vulva, vagina, cervix and pelvis, uterus, fallopian tubes, ovaries, pelvic peritoneum and pelvic connective tissue. Inflammation may be confined to a single site or may involve multiple organs. Mild cases are asymptomatic, while severe cases such as acute pelvic inflammatory disease can cause diffuse peritonitis, bacteremia, infectious shock, and even death. The anatomical and physiological and biochemical characteristics of the female reproductive tract have better natural defenses, which can lead to inflammation when the natural defenses are disrupted, or when the immune function of the body decreases, when endogenous flora changes or when exogenous pathogenic bacteria invade. Reproductive system inflammation classification: including lower reproductive tract infections: such as vulvodynia, vaginitis, cervicitis, pelvic inflammatory diseases; including endometritis, tubal inflammation, tubal ovarian inflammation, pelvic peritonitis and pelvic connective tissue inflammation, as well as reproductive tract tuberculosis and sexually transmitted diseases. Generally speaking, gynecological infections have three main characteristics: 1, the common pathogenic bacteria mainly from the normal parasitic flora of the reproductive tract; 2, anaerobic bacterial infections account for a large proportion; 3, mixed infections often occur, such as aerobic bacteria or parthenogenic aerobic bacteria and anaerobic bacteria co-exist and interact, resulting in treatment difficulties. The common pathogenic microorganisms that cause gynecological inflammation include a variety of microorganisms such as bacteria, viruses, fungi and protozoa. Bacteria: gonococci, gram-negative bacilli, Corynebacterium vaginalis, Duchene, Gardnerella, etc.; 2. Viruses: simple scarab virus, human papilloma virus, infectious soft spot virus, human immunodeficiency virus; 3. Fungi: Candida albicans; 4. Chlamydia: Chlamydia trachomatis; 5. Protozoa: Trichomonas vaginalis, etc. Non-specific vulvovaginitis: various pathogens invade the vulva can cause vulvovaginitis, non-specific vulvovaginitis is the most common. Diagnostic points 1, etiology vulvar infection such as Escherichia coli, vulvar impurity or foreign body stimulation such as abnormal vaginal secretions, urine, chemical fiber fabric underwear caused by non-specific inflammation. 2.History of diabetes mellitus, urinary and fecal repeated disease. 3, clinical manifestations vaginal discharge increased, vulvar skin phlegm itching, pain, burning sensation; local congestion, swelling, erosion, ulceration, eczema; chronic inflammation when the skin is thickened, rough, more cracked or even mossy-like changes; inguinal lymph node enlargement occasionally seen. Ancillary examinations 1, vaginal secretions for trichomonas and fungal examination, except for vulvovaginitis caused by atopic vaginitis. 2. Chlamydia, mycoplasma and gonococcal tests should be performed on cervical secretions. 3.Bacterial culture and drug sensitivity test of vaginal secretion if necessary. 4. Perform tissue biopsy if necessary for vulvar ulcers. 5.Diagnosis can be made initially based on clinical manifestations. Those with positive reaction belong to atopic vulvovaginitis. Treatment principles 1. Keep the vulva dry and avoid scratching. 2. Etiological treatment: use the appropriate treatment for different causes. 3, local treatment 0 .1% iodine attachment or 1:5000 potassium permanganate sitz bath, etc.