The incidence of congenital clubfoot accounts for approximately 1 in 1,000 surviving newborns, and bipedal deformity accounts for approximately 50%. There are two general theories for its pathogenesis: 1. it is believed that the persistent plantar flexion and pronation of the talus caused by the defect of the primitive germ in the talus, and the secondary soft tissue changes in multiple joints and muscle-tendon complex; 2. it is believed that the primary abnormality of the soft tissue within the neuromuscular unit causes secondary bony changes. Pathological changes: 3. The basic changes are plantarflexion, inversion and inversion deformity. Treatment: 1. Non-surgical treatment: In recent years, the ponseti cast technique has regained favor, and most medical centers now believe that most clubfoot can be treated with a ponseti cast without surgery. It has been reported that 90-98% of children with clubfoot are treated successfully with this method. (Our experience is that the earlier the treatment the better, the best age is within 1 week after birth, generally after 4-5 stages of plaster orthopedic treatment, and then percutaneous Achilles tendotomy). 2, surgical treatment, generally for children with very stiff clubfoot deformity, manipulation and plaster orthopedic failure. It mainly includes posterior medial release, extensive posterior medial release + posterior lateral release, Achilles tendon lengthening + posterior capsulotomy, etc.