What is sleep apnea hypoventilation syndrome? Some people snore very seriously in daily life, not only snoring loudly, but also always snoring without gasping for breath, and then snoring again after a loud sound ranging from ten to tens of seconds, and holding their breath again after a few snores, and their family members are always worried about the danger of him, and push him awake when they see him holding his breath too much. The patient is also drowsy during the day and always wants to sleep. This disease is called sleep apnea syndrome in medical science. Sleep apnea hypoventilation syndrome can have what clinical manifestations: 1, daytime drowsiness, weakness, fatigue, sleep after sleep can not be relieved, often dozing. 2, morning headache or dizziness, and feel dry mouth and bitterness. 3.Lack of concentration during the day, memory loss, and short temper. 4.Increased nocturnal urination, acid reflux, and heartburn.5 Symptoms such as decreased sexual function. Of course, not every patient has the above clinical characteristics, especially in mild cases, the patient himself is often unaware of it, his wife, children, friends often have the best opportunity to observe the patient’s apnea episodes, their observations and narratives can often provide valuable diagnostic clues. A large number of epidemiological surveys show that the prevalence of hypertension among snoring and sleep apnea patients can be more than 50%, which is much higher than 11%-12% of the population. And this prevalence is related to the severity of sleep apnea, that is, the more serious the sleep apnea, and the more obvious the trend of hypertension. Conversely, the prevalence of sleep apnea in people with hypertension is significantly higher than in the population, reaching 30-40%. Secondly, simultaneous sleep apnea monitoring and continuous blood pressure monitoring revealed that sleep apnea can cause an increase in blood pressure at night, and the increase in blood pressure is also closely related to the severity of sleep apnea. SAS not only affects the absolute level of blood pressure, but also changes the rhythm of blood pressure over 24 hours. SAS not only affects the absolute level of blood pressure, but can also alter the 24-hour rhythm of blood pressure, resulting in a 24-hour “non-spoon” change in blood pressure, with an increase rather than a decrease in blood pressure at night. Another characteristic that distinguishes SAS from other hypertensive patients is that their blood pressure is mostly highest in the morning, with headaches and dizziness evident in the early morning, making medication alone less effective. More direct evidence that sleep apnea causes hypertension is that after complete or significant relief from sleep apnea, hypertension also decreases significantly, or even reaches normal. Sleep apnea and angina, myocardial infarction angina, myocardial infarction is due to acute myocardial ischemia and hypoxia, sleep apnea caused by nocturnal hypoxemia, increased blood pressure, increased heart rate, hemodynamic changes can lead to cardiac ischemia, hypoxia, sleep apnea can aggravate or induce angina or myocardial infarction is not difficult to understand. Acute myocardial infarction is the result of prolonged ischemia and hypoxia of the heart, and is also closely related to sleep apnea syndrome. In 1995, in the world-famous Lancet medical journal, Swedish researchers reported ten cases of sleep apnea syndrome patients diagnosed with nocturnal angina pectoris, and after treatment After removal of sleep apnea, the angina disappeared and the number of cardiac ischemic episodes was significantly reduced on ambulatory electrocardiography. Studies of apnea and arrhythmias have concluded that elevated vagal tone and hypoxemia caused by sleep apnea are the main mechanisms leading to cardiac arrest. This cardiac arrest is in a sense a protective mechanism for the heart, which can reduce the work done by the heart by slowing it down to reduce the oxygen consumption of the heart and avoid further damage to the heart. This protective mechanism is extremely important especially in the case of severe hypoxia caused by sleep apnea. It is worth noting that if a pacemaker is simply installed without simultaneous treatment of sleep apnea, although it can eliminate cardiac arrest, it also eliminates this protective mechanism, and instead the heart becomes more severely hypoxic. Thus, for patients with recurrent cardiac arrest at night, before installing a pacemaker for treatment, it is important to think that it may be caused by sleep apnea and sleep breathing monitoring should be performed. Is breath-holding at night a heart attack? The sudden onset of breath-holding and panic during sleep at night, which is relieved by sitting and resting for a few moments after waking up, is medically known as “nocturnal sleep apnea” and “telangiectatic breathing”, and is a typical sign of cardiac insufficiency. Cardiac arrhythmias that occur at night can also manifest as panic, chest tightness, and a strong sense of fear. These symptoms are very similar to those of nocturnal sleep apnea, and because some medical personnel do not know much about sleep apnea, it is very easy to misdiagnose sleep apnea syndrome as heart disease. There have been many articles reporting that many patients with severe sleep apnea who have been misdiagnosed as heart disease for a long time have been treated well after polysomnography sleep apnea monitoring has confirmed the diagnosis of sleep apnea syndrome. If you have the above symptoms along with uneven snoring, you should also think about the possibility of sleep apnea and visit the relevant sleep breathing disorder treatment center.