If the passage of the pharynx narrows during sleep, the airflow will make a sound when it passes, which is snoring. Generally, fat people, people with loose throat muscles, people with small jaw deformities, and people with inflammation of the throat are most likely to snore. People who snore severely often have OSAHS (sleep apnea syndrome), which means that there is a pause in breathing or a decrease in ventilation during the whole process of sleep, and the oxygen in the blood is reduced, which means that the oxygen taken in throughout the night is less than that of normal people. OSAHS damage to the cardiovascular system: OSAHS is the most obvious damage to the cardiovascular system, OSAHS is an independent risk factor for hypertension, coronary heart disease, leading to angina, myocardial infarction, heart failure, arrhythmia and sudden death at night. It is now clear that OSAHS is an independent risk factor for hypertension in addition to obesity, age, genetics, and smoking. there is not only a correlation but also a causal relationship between OSAHS and hypertension. Epidemiological findings suggest that the prevalence of OSAHS among hypertensive patients is about 30%-50%, and the prevalence of hypertension among OSAHS patients is 50%-80%, while the prevalence of hypertension in the general population is usually 10%-20%. Fifty percent of patients with severe OSAHS have coronary artery disease, and nearly 30% of OSAHS patients develop myocardial ischemia during nighttime sleep, especially during the REM sleep phase. One study showed that OSAHS patients had a significantly higher chance of dying from myocardial infarction and stroke (37.5%) during the 5-year follow-up period. In addition, bradycardia occurs in 80% of patients with OSAHS, and the incidence of premature ventricular contractions ranges from 57% to 74%. second-degree AV block can occur in more than 10% of patients. Frequent premature beats can occur when there is a significant decrease in blood oxygen. Sleep apnea damage to cerebrovascular and central nervous system: OSAHS patients are prone to ischemic stroke at night due to cerebral arteriosclerosis, increased blood viscosity, and enhanced platelet aggregation during hypoxia, coupled with slow cerebral blood flow. Elevated blood pressure at night predisposes to cerebral hemorrhage. 96% of elderly OSAHS patients have varying degrees of dementia, which is thought to be related to apnea, severe hypoxemia resulting in damage to cerebral hemispheres, especially cortical and subcortical functions. Children can suffer from chronic sleep apnea, which affects mental development, slow reactions, and memory loss that interferes with learning. Shrieking, restlessness and restless legs syndrome can occur during sleep due to hypoxemia. It can even cause mania and depression: speech dysfunction, slurred speech, morning headache, daytime sleepiness, behavioral changes, and personality abnormalities. Repeated awakenings during sleep and tossing and turning at night also predispose to epilepsy. Patients with OSAHS have dysfunction of respiratory center and respiratory muscles, decreased pulmonary ventilation, and severe respiratory distress signs and symptoms, such as cyanosis, convulsions, pulmonary edema, hypoxemia and hypercapnia, and prolonged apnea, acute respiratory failure, or nocturnal asthma. Pulmonary function, pulmonary hypertension and blood oxygen and carbon dioxide levels are significantly correlated. Long-term pulmonary hypertension can cause right ventricular hypertrophy, which can lead to pulmonary heart disease. The effect of OSAHS on endocrine and sexual function: OSAHS patients have increased release of liver glycogen due to apnea, hypoxia, increased anaerobic enzymes of sugar, increased insulin antibodies, obesity and other relative deficiencies of pancreatic islets, which can cause disorders of glucose metabolism and reduced glucose tolerance, resulting in non-insulin-dependent diabetes. Snoring is a bedroom “tumor”, not only seriously affects the quality of sleep of couples, but also a neglected sex “killer”. The repeated occurrence of apnea during sleep prevents people from getting enough oxygen, and the blood oxygen needed for penile erection will be in short supply, reducing the production of nitric oxide enzyme that controls erection, resulting in slow erection, reduced erection hardness or too short erection duration, or even inability to erect causing sexual dysfunction. On the other hand, snoring often causes disgust and resentment to the other party, which affects the sexual life of the couple and even causes separation of the couple. V. The effect of OSAHS on pregnant women: Researchers in Europe and the United States have suggested that snoring by pregnant women may affect the fetus in the womb. Pregnant women who snore have a high chance of preeclampsia, and the blood pressure of pregnant women who snore severely will increase, and these pathophysiological changes will affect the development of the fetus, resulting in delayed or even non-development of the fetus. The effect of OSAHS on kidney function: 64% of OSAS patients have proteinuria, 80% of which are mainly clear protein. Long-term causes nephrotic syndrome. The effect of OSAHS on the quality of life and work: OSAHS patients have poor sleep quality, reduced effective sleep, hypoxia, and suppressed cortical function, resulting in drowsiness, fatigue, daytime sleepiness, and in severe cases, car accidents due to dozing off while driving, which can cause serious injuries to themselves and others and bring great harm to society. The incidence of car accidents is 7 times higher than that of drivers without OSAHS, and the death rate of car accidents caused by drowsiness accounts for 83% of traffic fatalities.