1.The active ingredient of cerebroside myostatin injection is ganglioside, which is abundant in the nervous system and is a component of nerve cell membrane. Ganglioside has high affinity with damaged parts, it is very easy to embed in the nerve tissue, promote the regeneration and repair of damaged nerve fibers or tissues, reduce the brain damage after hypoxia-ischemia, and can reduce the disability rate of HIE. 2.CytophosphatidylcholineCytophosphatidylcholine can promote lecithin synthesis, improve brain cell membranes, improve mitochondrial respiratory function and endoplasmic reticulum function, improve the function of the brainstem reticular upstream system, enhance the role of the cone system in inhibiting the extracorporeal system, improve cerebrovascular function and increase cerebral blood flow. Its prominent effects are to promote awakening, reduce cerebral edema, and improve cellular metabolism. Cytidylcholine, as a brain cell activator, is the activated form of choline and has an important role in the synthesis of lecithin. Lecithin is an indispensable component of nerve cells. When brain cells are damaged, phospholipids, especially lecithin, are degraded in brain tissues, and cytidylcholine can promote phospholipid synthesis, inhibit decomposition, reduce the production of free fatty acids and increase the level of diglycerides, and protect brain cells from the surface, especially for the reduction of apoptosis in delayed neuronal injury. 3.Brain activator is an aqueous solution of organ-specific amino acid mixture extracted from animal brain protein, which is a low molecular peptide composed of essential free amino acid combination, can pass the blood-brain barrier and directly enter the nerve cells of brain, acting on protein synthesis and affecting its metabolism, with the ability to promote human brain nerve cell protein synthesis and affect its respiratory chain, improve brain metabolic function, increase the utilization of glucose and oxygen in brain tissue. It can improve the utilization of oxygen, normalize the inhibited glucose transport, and improve the ability of brain tissue to resist hypoxia and toxic substances. 4.Monosialic acid hexose ganglioside (GM-1) ganglioside is a natural component of nerve cell membrane, a kind of glycosphingolipid, which can obviously protect nerve cells and promote the recovery of various enzyme activities of cell membrane. It has been shown that monosialic acid tetracosylated ganglioside can significantly protect neuronal injury and promote neural regeneration and reduce cell death around the lesion.GM-1 is a new drug for repairing the central nervous system after injury and is one of the most important gangliosides, which plays an important role in neuronal differentiation, growth, axoplasmic transport and regeneration, and has a protective effect on hypoxic-ischemic brain injury. GMl is an important ganglioside present in mammalian neural tissue and plays an important role in neuronal growth axoplasmic transport and regeneration, and is an antagonist of GMl glutamate receptor hyperactivation. It selectively inhibits a variety of pathophysiological changes caused by glutamate receptor overactivation, has significant scavenging of free radicals, reduces endothelin, prevents sustained brain tissue damage caused by strong spasm of blood vessels and microvessels in the ischemic area, reduces apoptosis, and thus has a cerebral protective effect against hypoxic-ischemic brain injury. In addition, GMl can effectively control brain edema, neurological necrosis, apoptosis and other pathological processes caused by hypoxia-ischemia in neonatal animals, inhibit excitatory amino acid elevation, protect brain cells and improve brain electrical activity. A large number of animal experiments have confirmed that GM1 can reduce the content of nitric oxide in the brain tissue of neonatal rats with hypoxic-ischemic brain injury, which has a certain protective effect on hypoxic-ischemic brain injury. GM1 can improve the learning and memory functions of neonatal rats after hypoxic-ischemic brain injury, and plays a certain role in promoting the higher functional activities of the brain. GM1 can pass the neonatal blood-brain barrier, prevent neuronal cell apoptosis, stabilize nerve cell membranes, maintain calcium homeostasis, and reduce post-hypoxic-ischemic brain injury, while promoting the repair and regeneration of damaged nerves.