Endometrial hyperplasia disorder is a benign lesion if it suggests simple hyperplasia, but if it is complex hyperplasia or atypical hyperplasia, it may be a precancerous stage and requires further treatment. The endometrium is the target organ of female hormones. Estrogen stimulates endometrial proliferation and progesterone stimulates endometrial secretion. If the estrogen level is too high or the progesterone level is too low to antagonize estrogen, the endometrium will be in a proliferative state for a long time, and in the process of proliferation, glandular cell carcinoma may be induced. The progression from simple hyperplasia to endometrial cancer takes several years, and the progression can be reversed through the use of drugs, such as progesterone supplementation, androgens to antagonize estrogen levels, targeted drugs to reduce estrogen, endometrial cauterization to reduce endometrial thickness, and hysterectomy for women without childbearing requirements to avoid further development of the disease.