Hepatitis B cirrhosis is actually a pathological concept, that is, a pathological state in which a proliferation of hepatic fibrous tissue is seen in the pathological histology, dividing and wrapping around the liver lobules to form pseudo lobules, due to the replication of the hepatitis B virus. Hepatitis B cirrhosis can be divided into early, middle and late stages. Early cirrhosis is only pathologically altered, and the morphology and function of the liver can be normal. In the middle and late stages, liver function abnormalities begin to appear, and the size of the liver can be reduced, often with complications. Functionally, the liver can be divided into compensated cirrhosis and decompensated cirrhosis. Compensated cirrhosis, as the name implies, has normal liver function and can even be reversed after treatment. In contrast, decompensated cirrhosis has abnormal liver function and is often accompanied by complications such as upper gastrointestinal bleeding, portal hypertension, ascites, encephalopathy, and hepatorenal syndrome.