If a patient with hyperthyroidism gets pregnant, whether the child can be wanted or not depends on the effects on the mother, on the fetus and on the pregnancy. For the mother: It may cause agitation, irritability, insomnia, palpitations, fatigue, fear of heat, excessive sweating, weight loss, hyperphagia, increased stool frequency or diarrhea. However, these symptoms are effectively treated with a small dosage of anti-thyroid medication, which may not cause serious consequences and have little effect on pregnancy. However, if there are obvious symptoms of hyperthyroidism, especially if there are serious symptoms such as thyroid crisis or thyrotoxic heart disease, it may have adverse effects on the mother and fetus, and it is necessary to consider carefully whether to terminate the pregnancy. For the fetus: maternal thyroid hormone receptor stimulating antibodies (TSAb) can stimulate the fetal thyroid gland through the placenta and cause fetal or neonatal hyperthyroidism. However, anti-thyroid medication during pregnancy, especially in the early stages of pregnancy, has a certain teratogenic risk, which requires patients to have regular maternity checkups and to terminate the pregnancy as soon as fetal abnormalities are detected. For pregnancy: The negative effects of hyperthyroidism on pregnancy are mainly miscarriage, premature birth, pre-eclampsia and placental abruption, etc. In serious cases, pregnancy should be terminated in time. Therefore, if the patient’s hyperthyroidism is not controlled, it is recommended not to get pregnant; if the patient is receiving anti-thyroid medication and the serum TT₃ and TT₄ reach the normal range, stop anti-thyroid medication or apply the minimum dose of anti-thyroid medication, you can get pregnant; if the patient with hyperthyroidism gets pregnant, it is necessary to consider whether to terminate the pregnancy by integrating various aspects such as the mother, to the fetus and the gestation period.