The diagnosis is generally not difficult in a typical convulsive state without other positive neurological signs. EEG electromyography and, if necessary, cranial CT and MRI of the mastoid cranium should be routinely performed to exclude mastoid and cranial disorders. Electrical stimulation of the supraorbital nerve on the affected side is characterized by simultaneous contraction of the orbicularis oculi and other muscles innervated by the facial nerve, while stimulation of the unilateral supraorbital nerve on the normal side or in other diseases only causes contraction of the unilateral supraorbital nerve innervated by the orbicularis oculi. Jannetta suggested that compression of the facial nerve at the pontocerebral root (REZ) by the small arteries that travel through it is the main cause of facial muscle spasm, and these vessels include the anterior inferior and posterior inferior cerebellar arteries as well as the thick varicose veins. After middle age these normal and neurocross-compressed vessels begin to harden, and long-term compression of the nerves can cause demyelination degeneration, causing crosstalk between nerve axons. Excitation becomes afferent from efferent to afferent, and there can be a large accumulation of abnormal potentials and release can cause facial muscle spasm attacks. This argument can also be used to explain the etiology of trigeminal neuralgia and glossopharyngeal neuralgia. However, in recent years, there are also many scholars who hold negative opinions because there are many people whose facial nerve is compressed by blood vessels and facial muscle spasm does not occur, while 20%-30% of patients with facial muscle spasm do not find the nerve compressed by blood vessels. Clinical manifestations]: The spasm begins with eyelid fluttering on one side, and gradually extends from the top down to half of the facial muscles, and only in severe cases can it involve the neck and shoulder muscles. This involuntary spasm, which cannot be controlled by oneself, can be triggered or aggravated by emotional stress and overexertion. It has been tested that this synchronous discharge of the facial muscles is 350 times per second, medical education. The actual fact is that the actual person is not a person who is not a person, but a person who is not a person. Generally, there is no seizure during sleep, but 11% of patients still twitch during sleep, affecting sleep. The seizures become more and more frequent, seriously affecting life and work. The muscle strength gradually decreases after a long period of illness, and in the advanced stage, it can develop into hemifacial paralysis and end.