Facial muscle spasm is a painless, intermittent, involuntary and irregular tonic contraction or clonic seizure of the facial muscles within the innervation of the same facial nerve. The symptoms are aggravated by emotions or nervousness, which seriously affects the life and work of patients. The pathogenesis of facial spasm is still unclear, and there are two main hypotheses in academic circles: 1. The “short circuit” hypothesis suggests that the facial nerve is compressed by blood vessels at the root exit zone (REZ) of the pontine sulcus, resulting in damage to the myelin sheath of the nerve there, which exposes the nerve fibers and forms an electrical conduction between the exposed nerve fibers. “The central theory suggests that long-term compression of the facial nerve by microvessels causes demyelination changes in the facial nerve, resulting in increased excitability of the facial nucleus accumbens and the formation of abnormal synapses between facial motor neurons, causing abnormal impulse delivery.