Facial spasm, also known as facial spasm (FS). It is a condition that manifests as involuntary twitching of half of the face; the twitches are paroxysmal, irregular, and of varying degrees, and may be aggravated by fatigue, mental stress, and voluntary movements. The onset of the disease mostly starts from the orbicularis oculi muscle and then involves the whole face. The disease mostly occurs after middle age and is commonly seen in women. The disease progresses slowly and eventually the affected facial muscles atrophy, called “paralytic facial palsy”, which seriously affects the quality of life of patients. Symptomatic treatment is generally used, but the results are not satisfactory. It is believed that the main cause of the disease is the pulsatile compression of the facial nerve root by the tortuous and prolonged intracranial artery, which leads to atrophy and degeneration of the myelin sheath at the site of nerve fiber compression, short-circuiting of the nerve action current, and when the excitation is superimposed to a certain degree, a burst of downward transmission is formed, causing facial twitching. Professor Jennatta of the United States has adopted “microvascular decompression”, which is the only ideal way to treat this disease from the etiology, curing the disease while completely preserving the function of the facial nerve without sequelae. It has been popularized in the medical field. The anatomical principle of surgery: usually the facial nerve is located anteromedially and the auditory nerve is located posteriorly and laterally, the former is gray and the latter is yellowish. Almost all arterial compression occurs within 5 mm of the facial nerve exiting the brainstem, mostly in the posterior inferior cerebellar artery, vertebral artery, anterior inferior cerebellar artery or its branches, and rarely in the veins. Most of the compressions are single-vessel compressions and a few are multi-vessel compressions. Since the relationship between the brain and blood vessels can be changed by lying on the side, any blood vessel 1 to 2 mm away from the facial nerve root is considered to be compressing the nerve. In typical facial spasms, there is often anterior and inferior compression of the facial nerve, while in atypical cases, there is posterior or superior compression. A microdebrider is used to separate the vessel from the nerve, and polyester wool is embedded between the vessel and the nerve. If the vein compression is difficult to separate, it can be cut off by electrocoagulation with bipolar electrocoagulation forceps.