In recent years, with the increasing incidence of various neurological diseases and the emergence of new diseases, the medical community will face a new round of challenges and breakthroughs. Facial spasm is a common disease in neurosurgery, mostly occurring in adults over 40 years of age, and the proportion of women is generally higher than that of men. What are the main manifestations of facial spasm, how should patients be diagnosed, and what tests are needed? The initial symptom of facial myasthenia is eyelid fluttering, which makes it easy to associate it with the folk saying “the left eye jumps for money, the right eye jumps for disaster”. Patients with brief eyelid fluttering can be given some symbolic meaning, but if the fluttering has been present for more than 3 months, it is important to be alert to the occurrence of facial myospasm and to go to the hospital as soon as possible for further detailed diagnosis. Facial myospasm is usually diagnosed by taking a medical history and observing symptoms. In addition to eyelid fluttering, facial myospasm gets worse day by day, and then moves to the corners of the mouth and, in severe cases, to the neck; the occurrence of facial myospasm is uncontrolled and irregular, with varying degrees, sometimes twitching for tens of seconds to return to normal, sometimes twitching for more than ten minutes without stopping, and a few patients also have headaches and tinnitus. Patients can also use CT or magnetic resonance imaging technology, thus providing an accurate basis for treatment. According to relevant information, the main causative factor of facial muscle spasm is the compression of the nerve root (facial nerve out of the brainstem area) by the surrounding blood vessels. Based on this principle, the compression of the facial nerve root by blood vessels can be relieved, and the patient can be cured of facial muscle spasm. Microvascular decompression is currently a standard procedure in the treatment of facial spasm, which effectively relieves the relevant blood vessels from the compression of the facial nerve root without damaging the function of the facial nerve, and then inserts a Tefflon spacer of the appropriate size after the two are fully free to complete the decompression of the nerve with ideal results.