Microvaxcular decompression (MVD) In 1962, Gar-dne et al. found intraoperatively that microvascular compression was closely associated with the onset of facial myoclonus. It is believed that facial myoclonus is a common reversible pathophysiological state caused by mild and continuous compression of the facial nerve by vascular structures in the pontocerebellar horn. Jannetta performed microvascular decompression for 78 cases of facial myasthenia from 1977 to 1992, and Kobdra in Japan reported 924 cases from 1984 to 1994, all of which achieved satisfactory results and accumulated rich experience. The current common method of microvascular decompression is: the patient lies supine with the head sideways to the healthy side. Local anesthesia with neuroleptic analgesia or general anesthesia. Internal incision in the posterior hairline; or transverse incision at the inferior end of the lower flat mastoid tip. The skin incision is 3~4 cm, and the skull is drilled below the intersection of the occipital mastoid suture or immediately below the mastoid emergence margin, followed by a bite to enlarge the bone window about 1.5~2 cm in diameter, lateral to the inner margin of the sigmoid sinus and superior to the lower margin of the transverse sinus. The dura mater was suspended after “⊥” shaped incision. The procedure was performed under cold light source illumination with 5x surgical magnification or microscope. Cerebrospinal fluid is drained, and a 0.5-cm-wide cerebral pressure plate is extended after the cerebellum is subducted to widen the exposure. Confirm the internal auditory foramen and lift the cerebellar vermis lobule posteriorly to reveal the vestibular nerve and the beginning segment of the facial nerve root. The arachnoid was sharply separated from the pontocerebellar region of the cerebellum with microscissors, and the surface of the facial nerve root initiation segment was carefully explored, and the compressed artery was carefully freed to separate it from the facial nerve root initiation segment. An appropriately sized piece of polyester or Teflon is cut and placed between the facial nerve and the vessel, and the polyester piece is wrapped around the facial nerve root for one week and secured with a silver clip at both ends. Or a muscle piece is placed between the artery and the nerve. After complete hemostasis, the incision is closed in layers. Barker (1995) reported 3.2% ipsilateral hearing loss and deafness and 7.4% facial muscle weakness as complications of this procedure. Currently, microvascular decompression is the surgical treatment of choice for facial myasthenia gravis. For unsatisfactory or ineffective results of pharmacological treatment of facial myospasm, microvascular decompression is recommended.