Tuberculous pericarditis treatment program: In the acute stage, bed rest should be provided and adequate nutrition should be ensured. The principle of anti-tuberculosis treatment is the same as active tuberculosis. When there is exudate, adrenocorticotropic hormone should be added promptly for 3 to 4 weeks, which can accelerate the absorption of exudate, reduce adhesions and prevent the development of constrictive pericarditis. If the medication is stopped too soon, pericardial exudate may recur, and another course of treatment will need to be repeated. If large amount of pericardial effusion affects respiration and cardiac function, pericardiocentesis should be performed to extract the fluid, and continuous pericardial drainage can be performed to relieve the symptoms of pericardial tamponade, and also reduce the pericardial adhesions and narrowing. As for constrictive pericarditis, once the diagnosis is confirmed, surgical treatment should be carried out. Only by stripping the adhesions and partially removing the pericardium can the heart be untied. Laboratory tests: 1. Tuberculin test Positive tuberculin test and the presence of tuberculosis foci in other parts of the body are helpful for the diagnosis. 25% of the patients have a negative tuberculin test. 2. pericardiocentesis fluid examination Similar to the exudate of tuberculous pleurisy, there may be a bloody pericardial effusion. Confirmation of diagnosis depends on finding tubercle bacilli in pericardial fluid, but the positive rate is low, 20%~50% of cases are positive for tubercle bacilli culture. Adenosine deaminase ADA in pericardial fluid is obviously increased to help diagnosis. 3, pericardial biopsy can be seen cheese-like granulation tissue, positive rate of 50% ~ 75%. Other auxiliary examination: 1, X-ray examination is very important to determine the pericardial effusion, effusion > 300 ~ 500 ml when the fluoroscopy of the cardiac shadow expanded into a pear-shaped or flask-shaped, the original arc disappeared, the heart beat weakened or disappeared. The shadow of the base of the heart widens and becomes spherical when lying on the back. The aorta becomes smaller and the superior vena cava widens. Wave counting photography is helpful for diagnosis. 2.Electrocardiography (1) S-T segment elevation: In the early stage (hours to days), except for the S-T segment of aVR and V1, the S-T segment of other leads is elevated, which is obvious in V5 and V6, bowing downward, and then gradually decreases to return to the equipotential line. (2) T-wave changes: early T-wave is upright, when S-T segment returns to baseline, T-wave is gradually flattened or inverted. After the inflammation subsides (within weeks to months), the T wave gradually returns to normal. If it becomes chronic, T-wave inversion may persist for a long time. (3) The QRS complex wave is seen to be under-voltage. (4) Sinus tachycardia. (5) A large amount of pericardial effusion may cause electrical alternation of P, QRS and T waves. And right bundle branch conduction block may appear. 3.Echocardiography 15ml of fluid can be detected. A dark area of fluid without echo is seen between the posterior wall of the left ventricle and the posterior pericardium; similarly, such a dark area may be present between the anterior wall of the right ventricle and the chest wall. Isotope scanning Intravenous injection of 131I-labeled clear protein or intravenous 99mTc for cardiac scanning, compared with the cardiac shadows on X-ray, can determine the presence or absence of effusion.