Postpartum stress urinary incontinence is secondary to postpartum stress urinary incontinence that occurs after pregnancy, mainly due to increased mobility of the bladder-urethral junction, defects in the anatomical structure of the urethra, and relaxation of the pelvic floor tissue in women during pregnancy. During pregnancy, the fetus gradually grows, its weight increases, the amniotic fluid increases, and the pressure on the muscles and fascia of the pelvic floor is gradually strengthened. The endocrine hormone level of pregnant women during pregnancy, their own changes, changes in the ligaments associated with the uterus, and the prolonged compression of the fetal previa during delivery, etc., cause the pelvic floor muscles and fascia to become overstretched and less elastic, often accompanied by partial rupture of the pelvic floor fibers. In addition, premature heavy physical labor, constipation, prolonged weight bearing and prolonged standing during the puerperium can cause or aggravate postpartum stress incontinence if the damaged pelvic floor muscles and fascia are not recovered in time and continue to be damaged. If the pelvic floor muscles and fascia are recovered in time by actively and positively insisting on postpartum rehabilitation exercises during the puerperium, taking good care of them and changing the bad routine to defecate in time, the pelvic floor muscles and fascia may recover to the unpregnant state during the puerperium. The postpartum pelvic floor exercises can reduce the occurrence of postpartum stress incontinence.