Cervical spondylosis is the culprit of many diseases
Spinal cord type cervical spondylosis
1.Sensory disorder of spinal cord type cervical spondylosis
(1) Sensory disorder in the trunk and normal sensation in the lower extremities, the trunk of such patients may have stripes of pain allergy, pain hyperalgesia and pain disappearance; there are also cases where pain allergy and pain hyperalgesia exist at the same time, often showing pain allergy in the upper stripes and pain hyperalgesia or pain disappearance in the lower stripes.
(2) There is sensory disorder in the trunk and sensory disorder in the lower extremities, often the sensory disorder of the trunk is connected with the sensory disorder of the lower extremities, and the sensory disorder of the lower extremities can be unilateral or bilateral.
(3) There is sensory disorder in the trunk and only local sensory disorder in the upper limb or lower limb, and the sensory disorder of the limb is either coat-like, lamellar, or only in the finger (toe) end.
2.Diagnosis of spinal cord type cervical spondylosis
(1) It mostly occurs in middle age or above, and the symptoms are initially numbness, weakness and upper motor neuron damage signs in the limbs or trunk. The symptoms are recurrent and progressively aggravated at the same time.
(2) Signs of neck pain and restricted movement in spinal cord type cervical spondylosis are not known as simple lower limb movement disorders (such as weakness, shaking, leg weakness or easy to fall); there are those who show simple lower limb sensory disorders (such as abnormal sensation in both feet and numbness in both lower limbs) and those who have sensory and motor disorders at times.
(3) Lateral symptoms. Sensory-motor disorders of the upper and lower extremities appear on the side, such as swelling of the right arm, along with pain and muscle tremors in the right lower back and right lower extremity.
(4) Crossed symptoms. Sensory or motor disorders appearing in one side and the contralateral lower extremity, such as numbness in the lateral upper extremity and pain in the contralateral lower extremity.
(5) Extremity symptoms. There are neurological disorders in the extremities, such as simple sensory disorders (e.g. numbness in the little toe of both feet and ulnar side of both hands); there are sensory and motor disorders in the extremities one after another in a short period of time, such as a patient who has numbness in the 4 or 5 fingers of the left hand on the next day and in the 4 or 5 fingers of the right hand on the third day after working with the head down for a long time, and feels numbness, weakness, difficulty in lifting the legs and unstable gait in both lower extremities on the fourth and fifth days.
(6) Head symptoms manifested as headache and dizziness.
(7) Sacral nerve symptoms. The symptoms are urination or defecation disorders, such as abnormal sensation of the head of the turtle, frequent urination, incomplete urination, lumbar and leg weakness, weakness in defecation or constipation, etc.
3.Signs of spinal cord type cervical spondylosis
Spinal cord type
1, unilateral compression of the spinal cord: When the spinal cord is unilaterally compressed, the typical or atypical Brown-Sepuard syndrome can appear. It is characterized by increased muscle tone, decreased muscle strength, hyperactive tendon reflexes, decreased superficial reflexes, and pathological reflexes in the ipsilateral limb below the level of the lesion; in severe cases, patellar clonus or ankle clonus can be induced. In addition, there are tactile and deep sensory deficits. The contralateral side is dominated by sensory disturbances, i.e., temperature and nociceptive disturbances. The distribution of the disorders does not correspond to the level of the lesion. Since the motor and proprioceptive tracts on the contralateral side are still normal, the motor function on that side is normal.
2.Bilateral compression of spinal cord: early symptoms are mainly sensory disorders or mainly motor disorders, and late symptoms are incomplete spastic paralysis with different degrees of upper motor neuron or nerve bundle damage, such as unfavorable activity, unstable walking, bedridden, and difficulty in breathing. There is increased muscle tone, decreased muscle strength, hyperactive tendon reflexes, and decreased superficial reflexes. Pathological reflexes are positive. The patient has a sensation of thoracic and lumbar fasciculations, and the plane of sensory alteration often does not correspond to the level of the lesion. Sometimes the plane and degree of sensory disturbance on the left and right sides do not match. In some cases, the planes of sensory disturbance are distributed in multiple segments, and in severe cases, there may be sphincter dysfunction.
(1) Mixed spinal cord and nerve root type. In addition to the symptoms and signs of spinal cord bundle involvement, there are also signs of cervical nerve roots, which are different from the neurogenic cervical spondylosis.
(2) Patients show varying degrees of spastic paraplegia, but the plane of sensory alteration is irregular and the function of sphincter force is less affected, which is different from transverse spinal cord injury.
(3) X-ray plain radiographs of spinal cord type cervical spondylosis can show that the plane of spinal cord compression basically conforms to that shown on plain radiographs, but in myelography, cervical spine with obvious bone spurs is not obstructed, while those without obvious bone spurs are clearly obstructed.
(4) CT and MRI scans or plus myelography with iodine oil can clearly diagnose or differentially diagnose cervical spondylosis caused by compression of the spinal cord, and the specific number and location of its spinal cord compression scale can be determined.
Thousands of forms – sympathetic cervical spondylosis
1.How to recognize the physiopathology of sympathetic cervical spondylosis?
Because patients with cervical spondylosis have degeneration of the intervertebral discs and reduced local stability, coupled with smaller intervertebral foramina, overlapping small joints, increased stress on the joint capsule and osteophytic factors, which cause a local traumatic reaction and provoke sympathetic nerve endings on the nerve roots and joint capsule and collateral ligament as well as the meningeal anterior branch in the spinal canal, producing a series of pathological reflex symptoms. There are two main reflex pathways.
(1) Spinal reflex
After the afferent fibers transmit information to the lateral anterior horn cells of the spinal cord, the reflex signal reaches the lower, middle and upper cervical segments via the preganglionic fibers of the lateral horn cells, where they alternate and then send out multiple groups of postganglionic fibers. The first group innervates the sweat glands and blood vessels of the face through the external carotid artery; the second group innervates the blood vessels of the brain and eyes and the sweat glands of the pupil and smooth muscle of the eyelids through the internal carotid artery; the third group innervates the blood vessels of the brainstem, cerebellum, temporal and occipital lobes of the brain, and inner ear through the vertebral artery; the fourth group is the postganglionic fibers from the three ganglia of the neck, which form the heart branch and control the heart rhythm.
(2) Brain a spinal cord reflex
The pathological stimulus of cervical spondylosis reaches the cerebral cortex through sympathetic afferent fibers and sensory fibers of the somatic nerves, and then signals are sent from the cortical cells to the cervical sympathetic ganglia for alternation through the middle and lower part of the optic thalamus, the midbrain periaqueduct, the red nucleus and the reticular structures below, and then postganglionic fibers are sent to the effector organs.
2.What are the clinical signs of sympathetic cervical spondylosis?
(1) Sympathetic excitation symptoms
(1) Head symptoms: headache or migraine, dullness, dizziness, occipital pain or posterior neck pain; however, these symptoms are not aggravated when the head moves.
②Facial symptoms: enlarged eye fissures, blurred vision, dilated pupils, swelling and pain in the eye sockets, dry eyes, and blinding of the eyes.
(iii) Cardiac symptoms: rapid heartbeat, disturbed heart rhythm, precordial pain and increased blood pressure.
④Peripheral vascular symptoms: cold and chilly limbs due to vascular spasm, low local temperature, or tingling sensation when the limbs are cold, or redness, swelling and pain aggravation. Symptoms of numbness in the neck, face and limbs are also seen, but the hyperalgesia is not distributed according to nerve segments.
⑤ Sweating disorder: manifests as excessive sweating. This phenomenon may be limited to one limb, head, neck, hands, feet, distal extremities, or half of the body.
(2) Sympathetic inhibition symptoms
Sympathetic inhibition is also vagal or parasympathetic excitation. Symptoms are dizziness, drooping eyelids, tearing and nasal congestion, bradycardia; low blood pressure, increased gastrointestinal motility, etc.
3.Which diseases should be differentiated from sympathetic cervical spondylosis?
(1) Inadequate coronary artery blood supply
The symptom is severe pain in the precordial region. Accompanied by chest tightness and shortness of breath, only one or both upper limbs ulnar reflex pain without upper fat cervical spinal nerve root irritation symptoms. There are abnormal changes in the electrocardiogram. The symptoms can be reduced when taking oil nitrate drugs.
(2) Neurosis
No X-ray changes of cervical spondylosis. No symptoms of nerve root and spinal cord compression, and the application of drug therapy has certain effect. However, long-term observation and repeated examination are required for differential diagnosis.
Severe damage to brain cells – vertebral artery type cervical spondylosis
1.What is the diagnosis diagnosis of vertebral artery type cervical spondylosis?
(1) Patients above middle age often suffer from vertigo, nausea, headache and vision loss due to changes in the head and neck position. In addition, the patient may have symptoms of nerve root irritation.
(2) At the onset of the disease, the patient’s neck movement is often restricted; the vertigo symptoms are caused by large neck rotation and posterior extension activities.
(3) When palpation examination of the posterior cervical region is performed, some patients may find displacement of the upper cervical vertebrae or other affected vertebrae, and swelling and pressure pain in the corresponding joint capsule.
(4) Pathological displacement of the affected vertebrae can be found in the frontal and lateral cervical spine and oblique X-ray plain film.
(5) In some patients, the sound of obstruction of vertebral artery blood flow can be heard in the upper clavicle of the affected side.
2.What are the typical symptoms of vertebral artery type cervical spondylosis?
(1) Vertigo
Vertigo is a common symptom in patients with vertebral artery carotid disease. Patients change their position due to neck extension or rotation to induce vertigo symptoms. The vertigo caused by ischemic lesion of the vestibular nerve nucleus usually lasts for a short period of time and disappears in a few seconds to a few minutes, and the patient may have mild disorientation and movement disorder at the onset, which is manifested as unstable walking or tilting to one side; the vertigo caused by ischemic lesion of the vestibular nerve nucleus is not accompanied by impaired consciousness. The vertigo caused by vestibular neuropathy is central vertigo; the vagal ischemic lesion is peripheral vertigo. Some patients feel nauseous and cannot raise their heads during acute onset. A few patients have symptoms such as diplopia, eye tremor, tinnitus and deafness.
Some patients can hear murmur of the vertebral artery due to distortion and negative blood flow on auscultation of the affected side of the clavicle. The thumb palpation of the back of the neck can feel the affected vertebrae rotating and shifting to one side, and there is obvious pressure pain in the spinous process and the joint of the shifted synapse.
(2) Headache
In patients with vertebral artery cervical spondylosis, headache and vertigo symptoms usually coexist at the onset. Occipital neuropathy is the main cause of headache. Because the occipital artery, a branch of the vertebral artery, supplies the occipital nerve, clinically, spasm of the vertebral artery causes ischemia of the occipital nerve and the occipital nerve, clinically, spasm of the vertebral artery causes ischemia of the occipital nerve and headache symptoms in the occipital nerve innervation area, which is intermittent throbbing pain, radiating from the back of one side of the neck to the occipital area and half of the head, with a burning sensation. In addition, the rhomboid muscle, which is innervated around the paravertebral nerve, can cause spasm of the rhomboid muscle after root lesion or trauma to the muscle, and the occipital nerve branch that penetrates from the rhomboid muscle can be squeezed to induce clinical symptoms, and when the atlantoaxial or pivotal spine is displaced, the occipital nerve that penetrates from it can be stimulated to cause headache.
3.How to recognize the physiopathology of vertebral artery type cervical spondylosis?
(1) Influence of bone spurs
When there are hyperplastic bone spurs above cervical 6, such as intervertebral disc lesions, the vertebral artery can be stimulated to spasm and its lumen can be narrowed by compression.
(2) Vascular variation
Under normal circumstances, the size of the vertebral artery canal is one-half that of the carotid artery. It is about 4mm, and the left and right arteries are equal to ensure the normal blood supply to the brain. In pathological conditions, if the vertebral artery is stimulated. Spasm or stenosis occurs, and symptoms of insufficient blood supply can occur.
(3) Lesions of blood vessels
The age of onset of cervical spondylosis and atherosclerosis is the same, both in middle age or above. The atherosclerotic atheromatous plaque is better in the vertebral artery from the branch of the subclavian artery that the first segment is seen between the two transverse processes; the third and fourth segments are more inferior. The basilar artery does not turn in the middle, and due to the convergence of blood and the formation of vortex at its beginning, end and branches, it is easy to damage the intima of the vessel and form atheroma. In this way, the atherosclerosis is easily affected by the cervical spine spurs after the atherosclerosis and produces symptoms of insufficient blood supply. In addition, when the intervertebral space becomes narrow because of the degeneration of the cervical disc, the cervical spine becomes shorter and the vertebral artery becomes relatively longer. When the vertebral artery has deformity or atherosclerosis, both the pulling of the neck activity and the impact of the blood flow can make the carotid artery longer and distort to affect the normal blood circulation.
4.What is the relationship between neck activity and the onset of vertebral artery type cervical spondylosis?
(1) Under normal circumstances, although turning the head can reduce the blood flow of one vertebral artery, the other vertebral artery can compensate and thus no symptoms will occur.
(i) ipsilateral reduction in blood supply: when to the right, the left subatlantoaxial articular surface slides forward and downward, and the right vertebral artery is twisted and narrowed.
(ii) Reduced contralateral blood supply: The vertebral artery is relatively fixed because it passes around the transverse atlantoaxial process and penetrates the dura mater through the foramen magnum. When the head is turned, the atlantoaxial spine also moves, pushing the contralateral vertebral artery out of the transverse foramen and obstructing blood flow.
Nerve root type cervical spondylosis
1. Clinical symptoms of neurogenic cervical spondylosis
(1) Pain in the neck and shoulder and numbness in the fingers
Pain is the main symptom of radiculopathy. In the acute stage, the patient’s head and neck can cause pain in the neck, shoulder and arm, or radiating pain in the upper limbs, often accompanied by numbness in the fingers, and the pain is heavy at night, affecting rest. A few patients use their hands to protect the affected area to prevent touching the neck from aggravating the symptoms. For patients with acute onset, attention should be paid to check whether the lesion is a cervical disc herniation. Patients with chronic onset tend to feel soreness in the neck or back of the shoulder, radicular pain in the upper limbs or numbness in the fingertips. In addition, there is also muscle weakness and muscle atrophy in the upper limbs. Some patients may have swelling of the affected limbs and dark red or pale skin. Wind and cold and strain can be the triggers for the onset of the disease, and some patients have a gradual onset without obvious triggers. Different lesions of the brachial plexus nerve roots cause different pain areas: cervical 5 nerve root lesion causes pain in the distribution area of the deltoid muscle; cervical 6 nerve root lesion radiates to the deltoid muscle and the radial side of the forearm and the thumb; cervical 7 nerve lesion radiates to the middle finger along the back of the upper arm and forearm; cervical 8 nerve root lesion radiates to the ring finger and little finger along the inner side of the upper arm and forearm; and nerve root lesion of the thoracic sternum causes pain in the inner side of the upper arm.
(2) Weakness of muscle strength
Weakness of upper limb muscles is a symptom caused by motor nerve damage, which is manifested by the patient’s difficulty in holding objects and some patients tend to fall off when holding objects. The skeletal muscles of the limbs are innervated by more than two nerves, and damage to individual nerves may result in mild muscle weakness, while involvement of the main nerve root may result in significant motor dysfunction.
(3) Tension in the neck muscles
Patients with cervical spondylosis often have symptoms of cervical tense plate. Stimulation of the cervical nerve roots may reflexively cause increased muscle tone or spasm in the innervated cervical and shoulder muscles. In the acute stage, examination can mostly show that the patient has tension on one or both sides of the back of the neck and localized pressure pain.
2.Diagnosis of neurogenic cervical spondylosis
Diagnosis of neurogenic cervical spondylosis is mainly based on the radicular symptoms in the patient’s complaints, physical signs such as tendon reflexes and pain changes in the upper limbs, palpation such as the position of the spinous process of the posterior neck and cervical spine X-ray, and most patients can be diagnosed in time.
(1) Symptoms and signs
Patients usually complain of pain in the neck, shoulder and arm and numbness of the fingers. In the acute stage, cervical muscle tension and limitation of neck movement may occur. Changes in neck position can trigger or aggravate the symptoms. Some patients show atrophy of the forearm and hand muscles. Intervertebral foraminal compression test is positive, and brachial plexus nerve pull test may be positive. Some patients have vertigo symptoms.
(2) Posterior cervical palpation examination
Most of the spinous processes of the affected vertebrae have pathological displacement and pressure pain, and the corresponding articular synovial joints are swollen with obvious pressure pain, which is an important diagnostic basis.
(3) Cervical spine X-ray plain film examination
By observing the patient’s frontal and lateral radiographs and oblique radiographs, in addition to finding osteophytes at the posterior edge of the vertebral body and the Luschka joint, the position of the cervical spine can be changed due to the displacement of the affected vertebra in some cases. The clinician can make a diagnosis of cervical spondylosis based on the symptoms and signs of cervical spondylosis.
3.The pathogenesis of cervical spine nerve root type
(1) Local irritation and compression factors of the nerve root
Because of degenerative pathological changes in the cervical spine, the patient’s cervical spine is prone to cervical spine osteophytes when the disease is of long duration, which is transformed into one of the factors of nerve root pathology. The dural sleeve of the bony nerve root in the Luschka joint or synovial part of the intervertebral foramen can be secondary to inflammatory reactions leading to increased local vascular permeability and impaired circulation, and secondary hypertrophy, adhesions and fibrotic lesions in the root sleeve. The nerve root may be distorted and deformed, which is an important factor in causing neurogenic cervical spondylosis.
(2) Displacement of the affected vertebra
Soft tissue strain such as the cervical intervertebral disc, synovial joint, joint capsule and its surrounding ligaments can often cause part of the cervical spine to lose its stability. Injury to the muscles of the neck and shoulders can cause a loss of balance in the muscles of the soft tissues bilaterally and cause the cervical vertebrae to shift. Clinically, it is common for the affected vertebrae to rotate and shift to one side, making the transverse diameter of the intervertebral foramen smaller, thus stimulating and compressing the nerve roots and producing symptoms.
(3) Insufficient blood supply to the nerve root artery
The cervical nerve root artery is a nutritive artery, which can become smaller in the transverse diameter of the intervertebral foramen due to the creation of myelomeningocele in the Luschka joint or rotation and posterior displacement of the affected vertebrae, causing compression of the anterior root artery in front of the nerve root, so that symptoms occur due to ischemic lesions of the nerve root.
(4) Anterior oblique muscle spasm in the neck
When the anterior oblique muscle contracts, the brachial plexus nerve and the subclavian vein between the anterior and middle oblique shifts are compressed, and the patient has symptoms of radiating pain from the shoulder to the upper extremity, numbness in the ulnar nerve innervation area, low skin temperature in the upper extremity, and limitation of neck movement due to muscle spasm.
Pathological changes arising from cervical spondylosis
The main pathological changes of cervical spondylosis are as follows
(1) Degeneration of the intervertebral disc; after the age of 30, the elasticity of the annulus fibrosus decreases and fissures may arise, and the cartilage plate also degenerates, especially the water content of the nucleus pulposus decreases and the elasticity also decreases, which may eventually lead to fibrosis and calcification. Degeneration of the entire intervertebral disc leads to thinning of the intervertebral space and narrowing of the intervertebral space can be seen on x-ray.
(2) Small joint changes: When the intervertebral space is narrowed, the force on the small joints increases, which can also cause damage over time, making the intervertebral foramen correspondingly smaller.
(3) Ligamentous changes: the ligamentum flavum is often thickened after middle age, and when it is significantly thickened, the spinal canal can become smaller and the spinal cord can be compressed posteriorly. The anterior longitudinal ligament and posterior longitudinal ligament also have small tears due to acute trauma or chronic exertion, followed by fibrosis or calcification, and calcification shadow can be shown on X-ray.
(4) Osteomalacia: Spinal space narrowing, hematoma calcification due to ligament injury, and excessive wear and tear of small joints can lead to osteomalacia, with cervical 5 and 6 as the preferred sites. Bone spurs can have both symptomatic and stabilizing effects, depending on the site of the osteophyte. In the case of osteophytes on the lateral posterior border of the cervical spine, the blood flow to the vertebral artery can be affected.
Cervical spondylosis can make the whole body sick
Cervical spondylosis causes widespread blood supply deficiencies in the brain, resulting in systemic disease.
There are more than 40 related diseases, so if they are treated separately, what is it!
The key to cure the disease is to recover from cervical spondylosis and improve the blood supply to the brain, and the hypertensive vascular reaction will disappear on its own, thus achieving the purpose of treating multiple diseases together and strengthening the brain and body.
Many patients are suffering from multiple diseases, I do not know why? They don’t eat well, don’t sleep well, look at this disease today, look at that disease tomorrow, don’t know what kind of drugs to eat, and seek medical treatment all over the place. Pessimistic and disappointed, listening to the fate of God. The suffering is unbearable!
Where is the cause of multiple diseases? Do we need to treat them separately?
The human being is a whole person, and there is an intrinsic connection between illnesses. One problem causes many problems, and by solving the main problem, other problems will be solved. For example, cervical spondylosis and the lack of blood supply to the brain caused by it can damage the brain and affect intelligence, and also lead to general illness and aging before it is too late. There are more than 40 kinds of diseases related to this, so if they are treated separately, they will be treated separately!
Cervical spondylosis and the lack of blood supply to the brain caused by it can be said to be “the source of all diseases”!
The key to treatment is to rehabilitate cervical spondylosis and improve cerebral blood supply, and the hypertensive vascular reaction will disappear on its own, thus achieving the purpose of treating multiple diseases together and strengthening the brain and body.
I. Cervical spondylosis: cervical spine misalignment, cervical spine osteophytes, soft tissue injury, cervical intervertebral disc herniation, “falling pillow”, “low head syndrome”, pain and numbness and weakness of the neck, shoulder, back and hand, etc.
Second, the brain is damaged by insufficient blood supply to the brain: the main performance of three aspects.
(a) Insufficient cerebral blood supply: headache and dizziness, vertigo, motion sickness, seasickness, postural hypotension, foreign body sensation in front of the eyes, tinnitus, dizziness and blurred vision from head-turning or squatting up, cerebral hemogram examination showing cerebral vascular spasm and insufficient blood supply to the vertebrobasilar artery, etc.
(ii) Brain dysfunction (excitement): insomnia and dreaminess, inattention, irritability, and mental variability.
(c), brain damage: memory loss, slow reaction, Parkinson’s sign, Alzheimer’s disease, brain atrophy, brain nerve damage, brain tissue congestion hemorrhagic edema, cerebral hemorrhage stroke, cerebral thrombosis, brain softening, cerebral infarction, etc.
Third, the “hypertensive vascular reaction” caused by the human body’s various systemic diseases will be more, it is estimated that not less than 50 kinds.
The relationship between cervical spondylosis and osteophytes
Osteomalacia can be said to be a normal physiological phenomenon. According to statistics, 45 to 50% of people over the age of 40 have osteophytes, and after the age of 60, more or less 80% of people will have osteophytes, and as we age, the cartilage of the joints gradually degenerates, the elasticity of the cells decreases, and the bones and joints are unknowingly worn down, especially the cervical and lumbar joints, which are more mobile. The damaged articular cartilage is difficult to repair when there are no blood vessels to supply nutrients. At this time, the blood circulation around the articular cartilage is more vigorous and compensatory cartilage growth occurs, which is the precursor of osteophytes. Over time, the cartilage is calcified, and this is called osteophytes, or bone spurs. In fact, as soon as the bone spur gradually adapts to the needs of joint movement, the spur will no longer grow.
X-rays of patients with cervical spondylosis show varying degrees of osteophytes or bone fragments in the cervical spine. Many scholars believe that these growths are not the primary cause of cervical spondylosis.
Physical examination of cervical spine bone specimens revealed that the common sites of hyperplasia were in the cervical spine4-6, with the highest rate of hyperplasia in the cervical spine5, at 83%, and the rate of hyperplasia in each site was based on the hook process. This is because the stress of the cervical spine during flexion and extension activities is concentrated in this area, thus making it prone to strain.
So, do these osteophytes, or bone spurs, need to be removed surgically?
There are many successful cases of cervical spondylosis due to the long-term existence of some patients’ bone spur and bone superfluous material growing into the vertebrae, which forms the clinical spinal cord type. The recurrence rate is still as high as one quarter or more. For this reason, surgery is not the best method, and the most active and effective prevention and treatment method is early detection and early treatment with several effective non-surgical methods.
A few questions about cervical spondylosis
1.Cervical spondylosis with neck, shoulder and back pain as the main manifestation is mostly light cervical spondylosis; the clinical manifestations of patients are often very serious, but because the diagnosis is easy and clear, targeted treatment can be effective in a very short period of time and the effect is stable.
2. Severe cervical spondylosis (vertebral artery cervical spondylosis, spinal cord cervical spondylosis) may have no obvious neck and shoulder pain, and some patients even have no symptoms in the neck and shoulder, but are mainly manifested by dizziness, headache, vertigo, tinnitus, deafness, insomnia or drowsiness, lower extremity dyskinesia, paralysis, and related quintuplegia symptoms, and are often diagnosed as neurology-related diseases; the reason for their misdiagnosis is also because the patients have no performance related to the back of the neck and shoulder. Some patients are diagnosed with cervical spondylosis, but because their local manifestations of the back of the neck and shoulder are mild or non-existent, their cervical spondylosis does not attract the attention of clinicians, and the diagnosis is still based on neurological diseases, and the treatment is still based on the treatment of neurological symptoms, while the effective treatment for cervical spondylosis is not applied in a timely manner, which makes many patients lose the opportunity for treatment; therefore, when certain diseases of neurology are diagnosed and cervical spondylosis is present at the same time, we will not be able to diagnose cervical spondylosis. Therefore, when certain neurological diseases are diagnosed and cervical spondylosis exists at the same time, we should attach great importance to the intrinsic connection between cervical spondylosis and these diseases, and pay attention to the treatment for the back of the neck and shoulder, so as to achieve the purpose of treating the cause and stabilizing and reversing the disease.
3, cervical spondylosis and related spinal diseases are closely related to a variety of chronic diseases; according to the research in the past 30 years, it is believed that cervical spine and thoracic-lumbosacral spine lesions are the source of multiple diseases closely related to a variety of chronic diseases throughout the body. Nowadays, research has found that many chronic cardiovascular diseases, many neurological and psychiatric diseases, and many diseases of the five senses are closely related to cervical spondylosis, and treatment for neck-related factors can lead to effective treatment of the diseases. In the related diseases, we will explain in detail what is now being studied.
Prevention of cervical spondylosis
(1) Pay attention to the correct posture of the head and neck, and insist on making forward leaning, backward leaning and left-right rotation 1-2 times a day for 10 minutes.
(2) Maintain a good sleeping posture, preferably with a soft metacarpal pillow to maintain the physiological curvature of the cervical spinous process forward.
(3) The height of the pillow should be about 10 cm is appropriate.
(4) The usual working position, so that neither the head is raised nor lowered to a comfortable position. When working for a long time, you should move your head and neck for an hour to make the neck ligament muscles get proper rest.
(5) Do not strain your neck when watching TV with your head up, it is best to keep the same level with your eyes. These measures can not only prevent cervical spondylosis, but also prevent the recurrence and aggravation of cervical spondylosis.